Plasminogen fragment K1-3 inhibits expression of adhesion molecules and experimental HCC recurrence in the liver

Int J Colorectal Dis. 2009 Jul;24(7):837-44. doi: 10.1007/s00384-009-0652-z. Epub 2009 Jan 27.

Abstract

Purpose: There is no established adjuvant or neo-adjuvant treatment to curb tumor recurrence of hepatocellular carcinoma (HCC). Recent data showed that angiostatic factors can inhibit tumor cell adhesion to the endothelium and therefore recurrence/metastasis. We tested a potential preventive, pre-operative strategy using plasminogen kringles 1-3 (K1-3) to overcome this hurdle.

Materials and methods: Effects of K1-3 on the intercellular cell adhesion molecule (ICAM) and vascular cell adhesion molecule (VCAM) expression was analyzed in vitro and in vivo on RNA and protein levels. Influence of K1-3 on HCC recurrence in the liver was analyzed in an orthotopic tumor model.

Results: K1-3 decreased ICAM expression in Hepa129 tumor cells and VCAM expression in SVEC4-10 endothelial cells in vitro. In vivo, ICAM was reduced in histological tumor sections. Preventive treatment with AdK1-3 inhibited experimental HCC recurrence and tumor growth in the liver.

Conclusions: We were able to show that K1-3 inhibits intrahepatic tumor recurrence. This novel aspect elucidates a possible approach to prevent HCC recurrence.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adenoviridae / genetics
  • Angiogenesis Inhibitors / pharmacology
  • Animals
  • Carcinoma, Hepatocellular / enzymology
  • Carcinoma, Hepatocellular / genetics
  • Carcinoma, Hepatocellular / metabolism*
  • Carcinoma, Hepatocellular / pathology
  • Cell Adhesion Molecules / genetics
  • Cell Adhesion Molecules / metabolism*
  • Cell Line, Tumor
  • Cell Proliferation / drug effects
  • Gene Expression Regulation, Neoplastic / drug effects
  • Genetic Vectors / genetics
  • Humans
  • Intercellular Adhesion Molecule-1 / genetics
  • Intercellular Adhesion Molecule-1 / metabolism
  • Kringles*
  • Liver Neoplasms, Experimental / enzymology
  • Liver Neoplasms, Experimental / genetics
  • Liver Neoplasms, Experimental / metabolism*
  • Liver Neoplasms, Experimental / pathology
  • Male
  • Matrix Metalloproteinase 9 / metabolism
  • Mice
  • Neoplasm Metastasis
  • Peptide Fragments / pharmacology*
  • Plasminogen / pharmacology*
  • Recurrence
  • Vascular Cell Adhesion Molecule-1 / genetics
  • Vascular Cell Adhesion Molecule-1 / metabolism

Substances

  • Angiogenesis Inhibitors
  • Cell Adhesion Molecules
  • Peptide Fragments
  • Vascular Cell Adhesion Molecule-1
  • Intercellular Adhesion Molecule-1
  • Plasminogen
  • Matrix Metalloproteinase 9