Effects of 1-bromopropane, a substitute for chlorofluorocarbons, on BDNF expression

Int Immunopharmacol. 2009 Apr;9(4):433-8. doi: 10.1016/j.intimp.2009.01.007. Epub 2009 Feb 2.

Abstract

1-Bromopropane (1-BP) has been widely used as an alternative to ozone-depleting chlorofluorocarbons in various industries. Although the neurotoxicity of 1-BP has been recently reported, there is little information about the effect of 1-BP on the cells in brain by experimental approach. Here we studied the effect of 1-BP on brain-derived neurotrophic factor (BDNF) expression in astrocytes in vitro. The BDNF mRNA level was remarkably decreased by 1-BP in a human astrocytoma cell line, U251, and in mouse primary astrocytes. The DNA-binding and specific reporter activity of cAMP response element-binding transcription factor (CREB), which is one of the key molecules regulating BDNF expression, were reduced by 1-BP in U251 and/or mouse primary astrocytes. Additionally, protein kinase A (PKA) activity was suppressed by 1-BP in U251. These results suggest that BDNF expression was affected by 1-BP through at least PKA.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Astrocytes / drug effects*
  • Astrocytes / metabolism
  • Brain-Derived Neurotrophic Factor / antagonists & inhibitors*
  • Brain-Derived Neurotrophic Factor / biosynthesis
  • Cell Line, Tumor
  • Cells, Cultured
  • Cyclic AMP Response Element-Binding Protein / antagonists & inhibitors
  • Cyclic AMP Response Element-Binding Protein / metabolism*
  • Cyclic AMP-Dependent Protein Kinases / antagonists & inhibitors
  • Cyclic AMP-Dependent Protein Kinases / metabolism*
  • Humans
  • Hydrocarbons, Brominated / pharmacology
  • Mice
  • Mice, Inbred C57BL

Substances

  • Brain-Derived Neurotrophic Factor
  • Cyclic AMP Response Element-Binding Protein
  • Hydrocarbons, Brominated
  • Cyclic AMP-Dependent Protein Kinases
  • 1-bromopropane