IGF-1 and G-CSF complement each other in BMSC migration towards infarcted myocardium in a novel in vitro model

Cell Biol Int. 2009 Jun;33(6):650-7. doi: 10.1016/j.cellbi.2009.03.002. Epub 2009 Mar 13.

Abstract

Stem cell capability enhanced with cytokine administration is a promising treatment for myocardial infarction. Bone marrow stem cells (BMSCs) were isolated from C57BL/6 mice (8-12 weeks old) expressing GFP and characterized with c-kit and CD34. Infarcted heart tissue fragments were placed into dishes with BMSCs and medium supplemented with G-CSF, SCF, IGF-1 or combinations thereof were given to the BMSC-infarcted myocardium in vitro model. The IGF-1-G-CSF group showed significantly higher migration (67.7% +/- 2.6) of c-kit(+) BMSCs towards the ischemic tissue and expressed MEF-2 (43.7% +/- 1.7). Of the single treatment groups, the G-CSF group demonstrated significantly higher migration of c-kit(+) BMSCs (60.5 +/- 2.7) with MEF-2 expression (38.7 +/- 1.4). IGF-1 complements G-CSF and was relatively more significant in its effects on BMSC migration and cardiac lineage commitment towards ischemic heart tissue.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Antigens, CD34 / metabolism
  • Bone Marrow Cells / physiology*
  • Cell Movement / drug effects*
  • Granulocyte Colony-Stimulating Factor / pharmacology*
  • Insulin-Like Growth Factor I / pharmacology*
  • Mice
  • Mice, Inbred C57BL
  • Myocardial Infarction / pathology
  • Myocardial Infarction / physiopathology*
  • Myocardium / pathology
  • Myogenic Regulatory Factors / metabolism
  • Proto-Oncogene Proteins c-kit / metabolism
  • Stem Cells / metabolism
  • Stem Cells / physiology*

Substances

  • Antigens, CD34
  • Myogenic Regulatory Factors
  • Granulocyte Colony-Stimulating Factor
  • Insulin-Like Growth Factor I
  • Proto-Oncogene Proteins c-kit