Prolonged illumination up-regulates arrestin and two guanylate cyclase activating proteins: a novel mechanism for light adaptation

J Physiol. 2009 Jun 1;587(Pt 11):2457-72. doi: 10.1113/jphysiol.2009.168609. Epub 2009 Mar 30.

Abstract

Light adaptation in vertebrate photoreceptors is mediated by multiple mechanisms, one of which could involve nuclear feedback and changes in gene expression. Therefore, we have investigated light adaptation-associated changes in gene expression using microarrays and real-time PCR in isolated photoreceptors, in cultured isolated retinas and in acutely isolated retinas. In all three preparations after 2 h of an exposure to a bright light, we observed an up-regulation of almost 100% of three genes, Sag, Guca1a and Guca1b, coding for proteins known to play a major role in phototransduction: arrestin, GCAP1 and GCAP2. No detectable up-regulation occurred for light exposures of less than 1 h. Functional in vivo electroretinographic tests show that a partial recovery of the dark current occurred 1-2 h after prolonged illumination with a steady light that initially caused a substantial suppression of the photoresponse. These observations demonstrate that prolonged illumination results in the up-regulation of genes coding for proteins involved in the phototransduction signalling cascade, possibly underlying a novel component of light adaptation occurring 1-2 h after the onset of a steady bright light.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adaptation, Ocular* / genetics
  • Animals
  • Arrestin / genetics
  • Arrestin / metabolism*
  • Cells, Cultured
  • Dark Adaptation
  • Electroretinography
  • Gene Expression Profiling / methods
  • Guanylate Cyclase-Activating Proteins / genetics
  • Guanylate Cyclase-Activating Proteins / metabolism*
  • Immunohistochemistry
  • Light*
  • Mice
  • Mice, Inbred C57BL
  • Oligonucleotide Array Sequence Analysis
  • Photic Stimulation
  • Photoreceptor Cells, Vertebrate / metabolism*
  • RNA, Messenger / metabolism
  • Rats
  • Rats, Long-Evans
  • Reverse Transcriptase Polymerase Chain Reaction
  • Time Factors
  • Up-Regulation
  • Vision, Ocular* / genetics

Substances

  • Arrestin
  • Guanylate Cyclase-Activating Proteins
  • Guca1a protein, mouse
  • Guca1b protein, mouse
  • RNA, Messenger