Differential expression of N-myc in phenotypically distinct subclones of a human neuroblastoma cell line

Cancer Res. 1991 Dec 1;51(23 Pt 1):6338-45.

Abstract

Neuroblastomas are malignant childhood neoplasms that arise from derivatives of the neural crest. We report the characterization of a new neuroblastoma cell line, designated NBL-W, derived from the primary tumor of a patient with stage IVS disease (S. L. Cohn, C. V. Herst, H. S. Maurer, and S. T. Rosen, J. Clin. Oncol., 5: 1441-1444, 1987) according to the criteria of Evans [A. E. Evans, G. J. D'Angio, and J. Randolf, Cancer (Phila.), 27: 374-378, 1971]. Neurite-bearing (N) and substrate-adherent (S) cell lines have been subcloned from the parent line. N and S cells can interconvert, and both cell types label with the neural crest cell surface marker antibody, HNK-1. Cells in the subcloned lines and in the parent line have been shown by Southern blot analysis to contain approximately 100 copies of the N-myc gene. Cytogenetic analysis shows a homogeneously staining region present on chromosome 19. Although these subclones are of identical genotype, the S cells express lower amounts of N-myc mRNA and protein as compared to the N cells. N cells express several neuronal proteins including the neurotransmitter-processing enzymes tyrosine hydroxylase and dopamine beta-hydroxylase, the neuronal intermediate filament proteins peripherin and NF66/alpha-internexin, and the neural cell adhesion molecule. S cells generally lack neuronal markers but express the mesenchymal intermediate filament protein vimentin, and a small subset of the S cells express glial fibrillary acidic protein. Some S cells were labeled weakly with neural cell adhesion molecule antibody; others were negative. S cells did not express the glial marker S-100 or a melanocyte marker, tyrosinase. Thus, S cells express the neural crest marker HNK-1 but do not express a set of antigens characteristic of any known cell type derived from the neural crest. These results are consistent with the suggestion that differential N-myc expression may be involved in the interconversion of N and S cells but indicate that the S cell phenotype need not represent a highly differentiated neural crest derivative.

Publication types

  • Research Support, Non-U.S. Gov't
  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Cell Differentiation / drug effects
  • Gene Expression Regulation, Neoplastic / genetics*
  • Genes, myc / genetics*
  • Humans
  • Intermediate Filaments / chemistry
  • Karyotyping
  • Nerve Growth Factors
  • Neural Crest / enzymology
  • Neuroblastoma / chemistry
  • Neuroblastoma / genetics*
  • Neuroblastoma / pathology
  • Phenotype
  • RNA, Messenger / analysis
  • RNA, Neoplasm / analysis
  • Tretinoin
  • Tumor Cells, Cultured / chemistry
  • Tumor Cells, Cultured / pathology
  • Vimentin / analysis

Substances

  • Nerve Growth Factors
  • RNA, Messenger
  • RNA, Neoplasm
  • Vimentin
  • Tretinoin