Contribution of IRF-3 mediated IFNbeta production to DNA vaccine dependent cellular immune responses

Vaccine. 2009 Mar 26;27(15):2144-9. doi: 10.1016/j.vaccine.2009.01.134. Epub 2009 Feb 10.

Abstract

The mechanism(s) by which DNA vaccines activate Ag-specific cellular immune responses is incompletely understood. Current findings indicate that IRF-3 plays an important role in this process. The IRF-3 dependent signaling pathway is triggered by the presence of intracytoplasmic DNA, and culminates in the production of type I IFNs. DNA vaccination of IRF-3 KO mice elicits a strong Ag-specific humoral response, yet CD4 and CD8 T cell responses (including the production of Th1, Th2 and Th 17 cytokines) are severely impaired. Although expression of the immunogenic protein encoded by the DNA vaccine was similar in IRF-3 KO vs wild type mice, antigen presentation was severely impaired in the KO animals. This defect was remedied by the co-delivery of an IFNbeta encoding plasmid. These findings suggest that the IRF-3/IFNbeta pathways are key to the induction of cellular immunity following DNA vaccination.

Publication types

  • Research Support, N.I.H., Intramural

MeSH terms

  • Adoptive Transfer
  • Animals
  • CD4-Positive T-Lymphocytes / immunology
  • CD8-Positive T-Lymphocytes / immunology
  • Cells, Cultured
  • Immunity, Cellular / immunology*
  • Interferon Regulatory Factor-3 / genetics*
  • Interferon-beta / biosynthesis*
  • Mice
  • Mice, Inbred C57BL
  • Mice, Knockout
  • Plasmids / immunology
  • Spleen / cytology
  • Spleen / immunology
  • Vaccines, DNA / immunology*

Substances

  • Interferon Regulatory Factor-3
  • Irf3 protein, mouse
  • Vaccines, DNA
  • Interferon-beta