Activation of relaxin-related receptors by short, linear peptides derived from a collagen-containing precursor

Ann N Y Acad Sci. 2009 Apr:1160:78-86. doi: 10.1111/j.1749-6632.2009.03827.x.

Abstract

In a screening effort based on algorithmic predictions for novel G-protein-coupled receptor (GPCR) peptide activators, we were able to identify and examine two novel peptides (P59 and P74) which are short, linear, and derived from a natural, previously unidentified precursor protein containing a collagen-like repeat. Both peptides seemed to show an apparent cAMP-related effect on CHO-K1 cells transiently transfected with either LGR7 or LGR8, usually after treatment with cAMP-generating forskolin, compared to the same cells treated with forskolin plus relaxin. This activation was not found for the relaxin-3 receptor (GPR135). In a set of follow-up experiments, both peptides were found to stimulate cAMP production, mostly upon initial stimulation of cAMP production by 5 micro M forskolin in cells transfected with either LGR7 or LGR8. In a dye-free cell impedance GPCR activation assay, we were able to show that these peptides were also able to activate a cellular response mediated by these receptors. Although untransfected CHO-K1 cells showed some cellular activation by both relaxin and at least one of our newly discovered peptides, both LGR7- and LGR8-transfected cells showed a stronger response, indicating stimulation of a cellular pathway through activation of these receptors. In conclusion, we were able to show that these newly discovered peptides, which have no similarity to any member of the relaxin-insulin-like peptide family, are potential ligands for the relaxin-related family of receptors and as such might serve as novel candidates for relaxin-related therapeutic indications. Both peptides are linear and were found to be active after being chemically synthesized.

MeSH terms

  • Animals
  • CHO Cells
  • Colforsin / pharmacology
  • Collagen / chemistry*
  • Cricetinae
  • Cricetulus
  • Cyclic AMP / metabolism
  • Models, Theoretical
  • Peptides / pharmacology*
  • Receptors, G-Protein-Coupled / genetics
  • Receptors, G-Protein-Coupled / metabolism*
  • Receptors, Peptide / genetics
  • Receptors, Peptide / metabolism*
  • Relaxin / pharmacology
  • Signal Transduction / drug effects
  • Signal Transduction / genetics
  • Transfection

Substances

  • Peptides
  • RXFP1 protein, human
  • RXFP2 protein, human
  • Receptors, G-Protein-Coupled
  • Receptors, Peptide
  • Colforsin
  • Relaxin
  • Collagen
  • Cyclic AMP