miR-210 promotes osteoblastic differentiation through inhibition of AcvR1b

FEBS Lett. 2009 Jul 7;583(13):2263-8. doi: 10.1016/j.febslet.2009.06.006. Epub 2009 Jun 9.

Abstract

Although microRNAs (miRNAs) are involved in many biological processes, the mechanisms whereby miRNAs regulate osteoblastic differentiation are poorly understood. Here, we found that BMP-4-induced osteoblastic differentiation of bone marrow-derived ST2 stromal cells was promoted and repressed after transfection of sense and antisense miR-210, respectively. A reporter assay demonstrated that the activin A receptor type 1B (AcvR1b) gene was a target for miR-210. Furthermore, inhibition of transforming growth factor-beta (TGF-beta)/activin signaling in ST2 cells with SB431542 promoted osteoblastic differentiation. We conclude that miR-210 acts as a positive regulator of osteoblastic differentiation by inhibiting the TGF-beta/activin signaling pathway through inhibition of AcvR1b.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Activin Receptors, Type I / antagonists & inhibitors*
  • Activin Receptors, Type I / metabolism
  • Animals
  • Bone Morphogenetic Protein 4 / metabolism
  • Cell Differentiation*
  • Cells, Cultured
  • Mice
  • MicroRNAs / metabolism*
  • Osteoblasts / cytology*
  • Osteoblasts / metabolism
  • Signal Transduction
  • Transfection
  • Transforming Growth Factor beta / metabolism

Substances

  • Bone Morphogenetic Protein 4
  • MicroRNAs
  • Transforming Growth Factor beta
  • Activin Receptors, Type I