Mitochondrial targeting of a catalase transgene product by plasmid liposomes increases radioresistance in vitro and in vivo

Radiat Res. 2009 May;171(5):588-95. doi: 10.1667/RR1424.1.

Abstract

To determine whether increased mitochondrially localized catalase was radioprotective, a human catalase transgene was cloned into a small pSVZeo plasmid and localized to the mitochondria of 32D cl 3 cells by adding the mitochondrial localization sequence of MnSOD (mt-catalase). The cell lines 32D-Cat and 32D-mt-Cat had increased catalase biochemical activity as confirmed by Western blot analysis compared to the 32D cl 3 parent cells. The MnSOD-overexpressing 32D cl 3 cell line, 2C6, had decreased baseline catalase activity that was increased in 2C6-Cat and 2C6-mt-Cat subclonal cell lines. 32D-mt-Cat cells were more radioresistant than 32D-Cat cells, but both were radioresistant relative to 32D cl 3 cells. 2C6-mt-Cat cells but not 2C6-Cat cells were radioresistant compared to 2C6 cells. Intratracheal injection of the mt-catalase-plasmid liposome complex (mt-Cat-PL) but not the catalase-plasmid liposome complex (Cat-PL) increased the resistance of C57BL/6NHsd female mice to 20 Gy thoracic irradiation compared to MnSOD-plasmid liposomes. Thus mitochondrially targeted overexpression of the catalase transgene is radioprotective in vitro and in vivo.

Publication types

  • Research Support, N.I.H., Extramural

MeSH terms

  • Animals
  • Catalase / genetics
  • Catalase / physiology*
  • Cell Line
  • Cell Survival / radiation effects
  • Female
  • Glutathione / analysis
  • Glutathione Peroxidase / analysis
  • Humans
  • Liposomes
  • Mice
  • Mice, Inbred C57BL
  • Mitochondria / enzymology*
  • Plasmids
  • Radiation Tolerance*
  • Superoxide Dismutase / physiology
  • Transgenes

Substances

  • Liposomes
  • Catalase
  • Glutathione Peroxidase
  • Superoxide Dismutase
  • Glutathione