Abstract
A small molecule inhibitor of MAP/ERK kinase (MEK) was effective against human breast cancer cells with a basal-like gene expression signature. Antitumor activity was limited by both feedback upregulation of phosphatidylinositol-3 kinase (PI3K)/AKT upon inhibition of MEK as well as loss of the phosphatase PTEN. Therefore, MEK inhibitors should preferably be investigated in combination with PI3K inhibitors in basal-like breast cancers.
MeSH terms
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Animals
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Antineoplastic Agents / pharmacology
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Biomarkers, Tumor / antagonists & inhibitors
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Biomarkers, Tumor / genetics
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Biomarkers, Tumor / metabolism
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Breast Neoplasms / drug therapy*
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Breast Neoplasms / metabolism
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Breast Neoplasms / pathology
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Cell Line, Tumor
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Enzyme Inhibitors / pharmacology*
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Female
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Humans
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MAP Kinase Kinase 1 / antagonists & inhibitors*
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MAP Kinase Kinase 1 / genetics
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MAP Kinase Kinase 1 / metabolism
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Mammary Neoplasms, Experimental / drug therapy
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Mammary Neoplasms, Experimental / metabolism
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Mammary Neoplasms, Experimental / pathology
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Mice
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Mice, Inbred Strains
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Mice, Nude
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Models, Biological
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Phosphatidylinositol 3-Kinases / genetics
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Phosphatidylinositol 3-Kinases / metabolism
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Phosphoinositide-3 Kinase Inhibitors*
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Xenograft Model Antitumor Assays*
Substances
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Antineoplastic Agents
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Biomarkers, Tumor
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Enzyme Inhibitors
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Phosphoinositide-3 Kinase Inhibitors
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MAP Kinase Kinase 1