Chemokine, vascular and therapeutic effects of combination Simvastatin and BMSC treatment of stroke

Neurobiol Dis. 2009 Oct;36(1):35-41. doi: 10.1016/j.nbd.2009.06.012. Epub 2009 Jul 8.

Abstract

We investigated the additive therapeutic effect of the combination treatment of stroke with sub-therapeutic doses of Simvastatin, a HMG-CoA reductase inhibitor, and bone marrow stromal cells (BMSCs). Rats were administered Simvastatin (0.5 mg/kg), BMSCs (1x10(6)) or combination of Simvastatin and BMSCs starting at 24 h after stroke. Combination treatment significantly improved neurological outcome, enhanced angiogenesis and arteriogenesis, and increased the number of engrafted-BMSCs in the ischemic brain. The number of engrafted-BMSCs and arteriogenesis was significantly correlated with functional outcome. Simvastatin significantly increased stromal cell-derived factor-1 (SDF1) expression in the ischemic brain and chemokine (CXC motif) receptor-4 (CXCR4) in BMSCs, and increased BMSC migration to RBMECs and astrocytes. Combination treatment of stroke upregulates the SDF1/CXCR4 axis and enhances BMSC migration into the ischemic brain, amplifies arteriogenesis and angiogenesis, and improves functional outcome after stroke.

Publication types

  • Research Support, N.I.H., Extramural
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Analysis of Variance
  • Animals
  • Blood Vessels / drug effects*
  • Blood Vessels / metabolism
  • Bone Marrow Cells / cytology
  • Bone Marrow Cells / physiology*
  • Bone Marrow Transplantation / methods
  • Bromodeoxyuridine / metabolism
  • Cell Movement / drug effects
  • Chemokine CXCL12 / metabolism
  • Disease Models, Animal
  • Gene Expression Regulation / drug effects
  • Gene Expression Regulation / physiology
  • Hydroxymethylglutaryl-CoA Reductase Inhibitors / pharmacology*
  • Infarction, Middle Cerebral Artery / therapy*
  • Male
  • Neovascularization, Physiologic / drug effects*
  • Neovascularization, Physiologic / physiology
  • Neurologic Examination / methods
  • Rats
  • Rats, Wistar
  • Receptors, CXCR4 / metabolism
  • Recovery of Function / drug effects
  • Recovery of Function / physiology
  • Simvastatin / pharmacology*
  • Stromal Cells / physiology

Substances

  • Chemokine CXCL12
  • Cxcr4 protein, rat
  • Hydroxymethylglutaryl-CoA Reductase Inhibitors
  • Receptors, CXCR4
  • Simvastatin
  • Bromodeoxyuridine