Feedback control of the EGFR signaling gradient: superposition of domain-splitting events in Drosophila oogenesis

Development. 2009 Sep;136(17):2903-11. doi: 10.1242/dev.039545. Epub 2009 Jul 29.

Abstract

The morphogenesis of structures with repeated functional units, such as body segments and appendages, depends on multi-domain patterns of cell signaling and gene expression. We demonstrate that during Drosophila oogenesis, the two-domain expression pattern of Broad, a transcription factor essential for the formation of the two respiratory eggshell appendages, is established by a single gradient of EGFR activation that induces both Broad and Pointed, which mediates repression of Broad. Two negative-feedback loops provided by the intracellular inhibitors of EGFR signaling, Kekkon-1 and Sprouty, control the number and position of Broad-expressing cells and in this way influence eggshell morphology. Later in oogenesis, the gradient of EGFR activation is split into two smaller domains in a process that depends on Argos, a secreted antagonist of EGFR signaling. In contrast to the previously proposed model of eggshell patterning, we show that the two-domain pattern of EGFR signaling is not essential for specifying the number of appendages. Thus, the processes that define the two-domain patterns of Broad and EGFR activation are distinct; their actions are separated in time and have different effects on eggshell morphology.

Publication types

  • Research Support, N.I.H., Extramural
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Body Patterning / physiology*
  • DNA-Binding Proteins / genetics
  • DNA-Binding Proteins / metabolism
  • Drosophila Proteins / genetics
  • Drosophila Proteins / metabolism*
  • Drosophila melanogaster / anatomy & histology
  • Drosophila melanogaster / embryology*
  • Drosophila melanogaster / physiology
  • Enzyme Activation
  • ErbB Receptors / genetics
  • ErbB Receptors / metabolism*
  • Eye Proteins / genetics
  • Eye Proteins / metabolism
  • Feedback, Physiological / physiology*
  • Female
  • MAP Kinase Signaling System / physiology*
  • Membrane Proteins / genetics
  • Membrane Proteins / metabolism
  • Mitogen-Activated Protein Kinases / metabolism
  • Nerve Tissue Proteins / genetics
  • Nerve Tissue Proteins / metabolism
  • Oogenesis / physiology*
  • Protein Tyrosine Phosphatases / genetics
  • Protein Tyrosine Phosphatases / metabolism
  • Proto-Oncogene Proteins / genetics
  • Proto-Oncogene Proteins / metabolism
  • Transcription Factors / genetics
  • Transcription Factors / metabolism*

Substances

  • Br protein, Drosophila
  • DNA-Binding Proteins
  • Drosophila Proteins
  • Eye Proteins
  • Membrane Proteins
  • Nerve Tissue Proteins
  • Proto-Oncogene Proteins
  • Transcription Factors
  • pnt protein, Drosophila
  • sty protein, Drosophila
  • aos protein, Drosophila
  • ErbB Receptors
  • Mitogen-Activated Protein Kinases
  • Kek1 protein, Drosophila
  • Protein Tyrosine Phosphatases