Differential effects of beta-endorphin on cAMP levels in human peripheral blood mononuclear cells

Brain Behav Immun. 1990 Sep;4(3):171-9. doi: 10.1016/0889-1591(90)90020-q.

Abstract

In the present paper we demonstrate that one of the early effects of the opioid peptide beta-endorphin on human peripheral blood mononuclear cells is the induction of a change in the intracellular cAMP level. However, the effect of beta-endorphin on cAMP levels is not uniform; increases as well as decreases in cAMP level are observed. It appears that beta-endorphin is a true modulator of intracellular cAMP level: the peptide will increase cAMP levels in cells with a low baseline level. In contrast, beta-endorphin tends to decrease cAMP levels is cells with a high cAMP concentration. Moreover, beta-endorphin modulates the rise in cAMP induced by beta-adrenergic activation. The effect of beta-endorphin on cAMP level correlates negatively with the magnitude of the change in cAMP level induced by beta-adrenergic activation.

MeSH terms

  • Adult
  • Cyclic AMP / metabolism*
  • Female
  • Humans
  • Immunity, Cellular / drug effects
  • Isoproterenol / antagonists & inhibitors
  • Isoproterenol / pharmacology
  • Leukocytes, Mononuclear / drug effects*
  • Leukocytes, Mononuclear / metabolism
  • Male
  • Middle Aged
  • Models, Biological
  • Neuroimmunomodulation
  • Peptide Fragments / pharmacology
  • Second Messenger Systems / drug effects*
  • Stress, Physiological / metabolism
  • beta-Endorphin / analogs & derivatives
  • beta-Endorphin / antagonists & inhibitors
  • beta-Endorphin / pharmacology*

Substances

  • Peptide Fragments
  • beta-endorphin (1-5)
  • beta-Endorphin
  • N-acetyl-beta-endorphin
  • Cyclic AMP
  • Isoproterenol