DNA demethylation of the perforin promoter in CD4(+) T cells from patients with subacute cutaneous lupus erythematosus

J Dermatol Sci. 2009 Oct;56(1):33-6. doi: 10.1016/j.jdermsci.2009.06.010. Epub 2009 Aug 3.

Abstract

Background: Recent evidence indicates that human lupus is an epigenetic disease characterized by impaired T cell DNA methylation. Perforin, a cytotoxic effector molecule, is overexpressed due to hypomethylation of its promoter regulatory elements in CD4(+) T cells from patients with systemic erythematosus lupus (SLE). However, it is unknown whether aberrant expression and methylation of perforin occur in CD4(+) T cells from patients with subacute cutaneous lupus erythematosus (SCLE).

Objective: We aimed to compare the perforin expression level and the methylation status of the perforin promoter region in CD4(+) T cells from SCLE patients and healthy controls.

Methods: We used real-time RT-PCR to compare the perforin mRNA levels, and Western-blot to compare perforin protein levels in CD4(+) and CD8(+) T cells from SCLE patients and healthy controls. Bisulfite sequencing was used to determine the methylation status of the perforin promoter region.

Results: Perforin is overexpressed in SCLE CD4(+) T cells. Demethylation of the perforin promoter region was seen in CD4(+) T cells from patients with SCLE.

Conclusions: DNA demethylation at the perforin locus contributes to perforin overexpression in SCLE CD4(+) T cells.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adult
  • CD4-Positive T-Lymphocytes / metabolism*
  • CD8-Positive T-Lymphocytes / metabolism*
  • DNA Methylation*
  • Female
  • Humans
  • Lupus Erythematosus, Cutaneous / genetics*
  • Male
  • Middle Aged
  • Perforin
  • Pore Forming Cytotoxic Proteins / genetics*
  • Promoter Regions, Genetic / genetics
  • Promoter Regions, Genetic / physiology
  • RNA, Messenger / metabolism

Substances

  • PRF1 protein, human
  • Pore Forming Cytotoxic Proteins
  • RNA, Messenger
  • Perforin