Dementia in SPG4 hereditary spastic paraplegia: clinical, genetic, and neuropathologic evidence

Neurology. 2009 Aug 4;73(5):378-84. doi: 10.1212/WNL.0b013e3181b04c6c.

Abstract

Background: Cognitive impairment and dementia has been reported in autosomal dominant hereditary spastic paraparesis (HSP) linked to the SPG4 locus. There has only been one postmortem examination described; not all accept that progressive cognitive decline is a feature of this disorder.

Objective: A family with SPG4-HSP known to have a deletion of exon 17 in the spastin gene (SPG4delEx17) was cognitively assessed over a 7-year period. The index family member died and a postmortem examination was performed.

Methods: Thirteen family members older than 40 years were clinically and cognitively assessed using the Cambridge Cognitive Assessment over a 7-year period. The presence of SPG4delEx17 was assessed; a neuropathologic examination of the brain of the index family member was performed.

Results: Cognitive decline occurred in 6 of the 13 family members and in all 4 older than 60 years. Two genetic deletions were identified: SPG4delEx17 in 12 of the 13 family members and a deletion of SPG6 (SPG6del) in 5. Eight individuals had the SPG4delEx17 deletion only; 4 had evidence of progressive cognitive impairment. Four family members had both SPG4delEx17 and SPG6del; 2 of these had cognitive impairment. One family member with the SPG6del alone had neither HSP nor cognitive impairment. The index case with both deletions died with dementia; the brain showed widespread ubiquitin positivity within the neocortex and white matter.

Conclusion: Cognitive decline and dementia is a feature of SPG4-HSP due to a deletion of exon 17 of the spastin gene.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adenosine Triphosphatases / genetics*
  • Adult
  • Age of Onset
  • Aged
  • Brain / metabolism
  • Brain / pathology
  • Brain / physiopathology
  • Cognition Disorders / diagnosis
  • Cognition Disorders / genetics
  • Cognition Disorders / physiopathology
  • DNA Mutational Analysis
  • Dementia / diagnosis*
  • Dementia / genetics*
  • Dementia / physiopathology
  • Disability Evaluation
  • Female
  • Gene Deletion
  • Gene Frequency / genetics
  • Genetic Markers / genetics
  • Genetic Predisposition to Disease / genetics*
  • Genetic Testing
  • Genotype
  • Humans
  • Inclusion Bodies / metabolism
  • Inclusion Bodies / pathology
  • Inheritance Patterns
  • Longitudinal Studies
  • Male
  • Membrane Proteins / genetics
  • Middle Aged
  • Neuropsychological Tests
  • Pedigree
  • Spastic Paraplegia, Hereditary / complications*
  • Spastic Paraplegia, Hereditary / genetics*
  • Spastin
  • Ubiquitin / analysis
  • Ubiquitin / metabolism

Substances

  • Genetic Markers
  • Membrane Proteins
  • NIPA1 protein, human
  • Ubiquitin
  • Adenosine Triphosphatases
  • Spastin
  • SPAST protein, human