Oxidative stress induced by morphine in brain of rats fed with a protein deficient diet

Hum Exp Toxicol. 2009 Sep;28(9):577-82. doi: 10.1177/0960327109102798. Epub 2009 Sep 10.

Abstract

The objective of the study is to determine the damage by oxidative stress induced by morphine in brain of rats fed with a protein-deficient diet. Twenty-eight malnourished male Wistar rats, 30 days old, were used in the study. The animals were divided into four groups of 7 rats per group. Group I received NaCl and the groups II; III and IV intraperitoneally received 3, 6 and 12 mg/kg of morphine sulphate, respectively, in a single dose. Animals were sacrificed and the levels of glutathione (GSH), dopamine, tryptophan and 5-hydroxyindole-3-acetic acid (5-HIAA) as well as, Na(+)/K(+) ATPase and total ATPase activity in the brain were measured. Tryptophan levels and Na(+)/K( +) ATPase activity showed non-significant changes in the experimental group. Levels of 5-HIAA decreased significantly (p = .03) in animals that received 12 mg/kg of morphine and in animals that received 3 mg/kg, levels of GSH and dopamine were found to have a significant decrease (p < .05), but a significant increase in the group that received 12 mg/kg of morphine (p < .05). Total ATPase activity increased significantly in the groups that received 3 mg/kg (p = .015) and 6 mg/kg (p = .0001) of morphine. The results show that malnutrition induces changes in cellular regulation and biochemical responses to oxidative stress caused by morphine sulphate.

MeSH terms

  • Analgesics, Opioid / adverse effects*
  • Animals
  • Antioxidants / metabolism
  • Biomarkers / metabolism
  • Brain / drug effects*
  • Brain / enzymology
  • Brain / metabolism
  • Dose-Response Relationship, Drug
  • Male
  • Morphine / adverse effects*
  • Oxidative Stress / drug effects*
  • Protein Deficiency / metabolism*
  • Rats
  • Rats, Wistar

Substances

  • Analgesics, Opioid
  • Antioxidants
  • Biomarkers
  • Morphine