[The effect of growth hormone-releasing factor (GRF) on secretion of insulin, glucagon and somatostatin from perfused rat pancreas]

Nihon Naibunpi Gakkai Zasshi. 1990 May 20;66(5):557-71. doi: 10.1507/endocrine1927.66.5_557.
[Article in Japanese]

Abstract

To examine the effects of growth hormone-releasing factor (GRF) on islet hormone release, rat pancreas was perfused. rhGRF at the concentration of 10(-7) M or more enhanced insulin secretion stimulated by 16.7 mM glucose, hpGRF slightly enhanced insulin secretion as well. The insulin secretion induced by 10(-6) M rhGRF was completely inhibited by 10(-6) M propranolol. rhGRF at the concentration of 10(-8) M or more stimulated glucagon secretion even in the presence of 16.7 mM glucose. The glucagon secretion stimulated by 10(-6) M rhGRF was inhibited in the early period but increased thereafter by 10(-6) M propranolol. 10(-6) M rhGRF slightly stimulated glucagon secretion in the presence of 16.7 mM glucose when STZ diabetic rat pancreas was perfused. rhGRF at the concentration of 10(-6) M enhanced somatostatin secretion stimulated with 16.7 mM glucose. We concluded that rhGRF stimulated insulin, glucagon and somatostatin secretion and the insulin secretion was inhibited by beta-blocker. hpGRF stimulated insulin and glucagon secretion as well.

MeSH terms

  • Animals
  • Diabetes Mellitus, Experimental / metabolism
  • Glucagon / metabolism*
  • Growth Hormone-Releasing Hormone / antagonists & inhibitors
  • Growth Hormone-Releasing Hormone / pharmacology*
  • Humans
  • In Vitro Techniques
  • Insulin / metabolism*
  • Insulin Secretion
  • Islets of Langerhans / drug effects*
  • Islets of Langerhans / metabolism
  • Male
  • Pancreas
  • Perfusion
  • Propranolol / pharmacology
  • Rats
  • Rats, Inbred Strains
  • Somatostatin / metabolism*

Substances

  • Insulin
  • Somatostatin
  • Glucagon
  • Growth Hormone-Releasing Hormone
  • Propranolol