Improved survival and reduced vascular permeability by eliminating or blocking 12/15-lipoxygenase in mouse models of acute lung injury (ALI)

J Immunol. 2009 Oct 1;183(7):4715-22. doi: 10.4049/jimmunol.0802592. Epub 2009 Sep 14.

Abstract

Acute lung injury (ALI) is a prevalent disease associated with high mortality. 12/15-lipoxygenase (12/15-LO) is an enzyme producing 12-hydroxyeicosatetraenoic acid (HETE) and 15-HETE from arachidonic acid. To test whether 12/15-LO is involved in increasing vascular permeability in the lung, we investigated the role of 12/15-LO in murine models of LPS-induced pulmonary inflammation and clinically relevant acid-induced ALI. The vascular permeability increase upon LPS inhalation was abolished in Alox15(-/-) mice lacking 12/15-LO and in wild-type mice after pharmacological blockade of 12/15-LO. Alox15(-/-) mice also showed improved gas exchange, reduced permeability increase, and prolonged survival in the acid-induced ALI model. Bone marrow chimeras and reconstitution experiments revealed that 12-HETE produced by hematopoietic cells regulates vascular permeability through a CXCR2-dependent mechanism. Our findings suggest that 12/15-LO-derived 12-HETE is a key mediator of vascular permeability in acute lung injury.

Publication types

  • Research Support, N.I.H., Extramural
  • Research Support, Non-U.S. Gov't

MeSH terms

  • 12-Hydroxy-5,8,10,14-eicosatetraenoic Acid / physiology
  • Acute Lung Injury / enzymology*
  • Acute Lung Injury / immunology
  • Acute Lung Injury / mortality*
  • Acute Lung Injury / pathology
  • Animals
  • Arachidonate 12-Lipoxygenase / deficiency*
  • Arachidonate 12-Lipoxygenase / genetics
  • Arachidonate 15-Lipoxygenase / deficiency*
  • Arachidonate 15-Lipoxygenase / genetics
  • Caffeic Acids / administration & dosage
  • Capillary Permeability / genetics
  • Capillary Permeability / immunology*
  • Cells, Cultured
  • Disease Models, Animal
  • Humans
  • Inflammation Mediators / administration & dosage
  • Lipopolysaccharides / administration & dosage
  • Lipoxygenase Inhibitors*
  • Mice
  • Mice, Inbred BALB C
  • Mice, Inbred C57BL
  • Mice, Knockout
  • Microcirculation / genetics
  • Microcirculation / immunology
  • Multienzyme Complexes / antagonists & inhibitors*
  • Multienzyme Complexes / deficiency*
  • Multienzyme Complexes / genetics
  • Survival Analysis

Substances

  • Caffeic Acids
  • Inflammation Mediators
  • Lipopolysaccharides
  • Lipoxygenase Inhibitors
  • Multienzyme Complexes
  • cinnamyl-3,4-dihydroxycyanocinnamate
  • 12-Hydroxy-5,8,10,14-eicosatetraenoic Acid
  • Alox15 protein, mouse
  • Arachidonate 12-Lipoxygenase
  • Arachidonate 15-Lipoxygenase