Abstract
In this issue of Molecular Cell, Fabian et al. (2009) demonstrate that in cell-free extracts from mouse Krebs-2 ascites, microRNA-mediated translational repression precedes target mRNA deadenylation, and identify GW182, PABP, and deadenylase subunits CAF1 and CCR4 as factors required for deadenylation.
MeSH terms
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Animals
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Argonaute Proteins
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Ascites / genetics
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Ascites / metabolism
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Autoantigens / metabolism
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Binding Sites
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Carcinoma, Krebs 2 / genetics
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Carcinoma, Krebs 2 / metabolism
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Eukaryotic Initiation Factor-2 / metabolism
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Eukaryotic Initiation Factor-4G / metabolism
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Exoribonucleases
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Gene Silencing*
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Humans
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Kinetics
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Mice
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MicroRNAs / metabolism*
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Poly(A)-Binding Proteins / genetics
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Poly(A)-Binding Proteins / metabolism*
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Protein Biosynthesis
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Protein Structure, Tertiary
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Proteins / genetics
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Proteins / metabolism*
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RNA Processing, Post-Transcriptional*
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RNA Stability
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RNA, Messenger / metabolism*
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RNA-Induced Silencing Complex / genetics
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RNA-Induced Silencing Complex / metabolism*
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Receptors, CCR4 / metabolism
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Repressor Proteins
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Ribonucleases
Substances
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Ago2 protein, mouse
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Argonaute Proteins
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Autoantigens
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Ccr4 protein, mouse
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Eukaryotic Initiation Factor-2
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Eukaryotic Initiation Factor-4G
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MicroRNAs
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Poly(A)-Binding Proteins
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Proteins
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RNA, Messenger
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RNA-Induced Silencing Complex
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Receptors, CCR4
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Repressor Proteins
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mirnlet7 microRNA, mouse
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Cnot7 protein, mouse
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Exoribonucleases
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Ribonucleases