Polyamines regulate c-Myc translation through Chk2-dependent HuR phosphorylation

Mol Biol Cell. 2009 Dec;20(23):4885-98. doi: 10.1091/mbc.e09-07-0550. Epub 2009 Oct 7.

Abstract

All mammalian cells depend on polyamines for normal growth and proliferation, but the exact roles of polyamines at the molecular level remain largely unknown. The RNA-binding protein HuR modulates the stability and translation of many target mRNAs. Here, we show that in rat intestinal epithelial cells (IECs), polyamines enhanced HuR association with the 3'-untranslated region of the c-Myc mRNA by increasing HuR phosphorylation by Chk2, in turn promoting c-Myc translation. Depletion of cellular polyamines inhibited Chk2 and reduced the affinity of HuR for c-Myc mRNA; these effects were completely reversed by addition of the polyamine putrescine or by Chk2 overexpression. In cells with high content of cellular polyamines, HuR silencing or Chk2 silencing reduced c-Myc translation and c-Myc expression levels. Our findings demonstrate that polyamines regulate c-Myc translation in IECs through HuR phosphorylation by Chk2 and provide new insight into the molecular functions of cellular polyamines.

Publication types

  • Research Support, N.I.H., Extramural
  • Research Support, U.S. Gov't, Non-P.H.S.

MeSH terms

  • 3' Untranslated Regions
  • Animals
  • Antigens, Surface / genetics
  • Antigens, Surface / metabolism*
  • Base Sequence
  • Checkpoint Kinase 2
  • ELAV Proteins
  • ELAV-Like Protein 1
  • Epithelial Cells / cytology
  • Epithelial Cells / drug effects
  • Epithelial Cells / physiology
  • Gene Expression Regulation / drug effects*
  • Gene Silencing
  • Intestinal Mucosa / cytology
  • Molecular Sequence Data
  • Phosphorylation
  • Point Mutation
  • Polyamines / metabolism*
  • Polyamines / pharmacology
  • Polyribosomes / chemistry
  • Polyribosomes / metabolism
  • Protein Biosynthesis*
  • Protein Serine-Threonine Kinases / genetics
  • Protein Serine-Threonine Kinases / metabolism*
  • Proto-Oncogene Proteins c-myc / genetics
  • Proto-Oncogene Proteins c-myc / metabolism*
  • RNA, Messenger / genetics
  • RNA, Messenger / metabolism
  • RNA-Binding Proteins / genetics
  • RNA-Binding Proteins / metabolism*
  • Rats

Substances

  • 3' Untranslated Regions
  • Antigens, Surface
  • ELAV Proteins
  • ELAV-Like Protein 1
  • ELAVL1 protein, human
  • Polyamines
  • Proto-Oncogene Proteins c-myc
  • RNA, Messenger
  • RNA-Binding Proteins
  • Checkpoint Kinase 2
  • CHEK2 protein, human
  • Chek2 protein, rat
  • Protein Serine-Threonine Kinases