[Construction and expression of PRL-3 plasmid with C104S point mutation and CAAX deletion]

Beijing Da Xue Xue Bao Yi Xue Ban. 2009 Oct 18;41(5):516-20.
[Article in Chinese]

Abstract

Objective: To construct PRL-3 gene C104S point mutation and CAAX deletion mutants: pcDNA3-myc-PRL-3 (C104S), pEGFP-PRL-3 (C104S), pcDNA3-myc-PRL-3 (DeltaCAAX) and pEGFP-PRL-3 (DeltaCAAX), and express these plasmids in eukaryotic cells.

Methods: Recombinant plasmids were mutated with pcDNA3-myc-PRL-3 plasmid as template and specific primers. Mutants were identified by restriction enzyme digestion and DNA sequencing. Then these recombinant plasmids were transfected into LoVo cells. The expression of fusion proteins were detected by western blotting and the localization of fusion proteins were examined by GPF fluorescence labelling.

Results: The mutants were successfully constructed and expressed in eukaryotic cells. PRL-3 (DeltaCAAX) relocates from plasma membrane/early endosome to the cytoplasm and/or nucleus, which provides a structural insight of PRL-3 protein.

Conclusion: Construction of eukaryotic expression recombinant plasmids of PRL-3 gene C104S point mutation and CAAX deletion mutants provide a useful tool for the study of PRL-3's role in cancer.

Publication types

  • English Abstract
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Base Sequence
  • Cell Line, Tumor
  • Colonic Neoplasms / pathology
  • Humans
  • Molecular Sequence Data
  • Neoplasm Proteins / genetics*
  • Plasmids / genetics*
  • Point Mutation*
  • Protein Tyrosine Phosphatases / genetics*
  • Proto-Oncogene Proteins c-myc / genetics
  • Recombinant Fusion Proteins / biosynthesis
  • Recombinant Fusion Proteins / genetics
  • Sequence Deletion*
  • Transfection

Substances

  • MYC protein, human
  • Neoplasm Proteins
  • Proto-Oncogene Proteins c-myc
  • Recombinant Fusion Proteins
  • PTP4A3 protein, human
  • Protein Tyrosine Phosphatases