Retrovirus delivered neurotrophin-3 promotes survival, proliferation and neuronal differentiation of human fetal neural stem cells in vitro

Brain Res Bull. 2008 Oct 22;77(4):158-64. doi: 10.1016/j.brainresbull.2008.02.037. Epub 2008 Apr 15.

Abstract

Poor survival and insufficient neuronal differentiation are the main obstacles to neural stem cell (NSC) transplantation therapy. Genetic modification of NSCs with neurotrophins is considered a promising approach to overcome these difficulties. In this study, the effects on survival, proliferation and neuronal differentiation of human fetal NSCs (hfNSCs) were observed after infection by a neurotrophin-3 (NT-3) recombinant retrovirus. The hfNSCs, from 12-week human fetal brains formed neurospheres, expressed the stem cell marker nestin and differentiated into the three main cell types of the nervous system. NT-3 recombinant retrovirus (Retro-NT-3) infected hfNSCs efficiently expressed NT-3 gene for at least 8 weeks, presented an accelerated proliferation, and therefore produced an increased number of neurospheres and after differentiation in vitro, contained a higher percentage of neuronal cells. Eight weeks after infection, 37.9+/-4.2% of hfNSCs in the Retro-NT-3 infection group expressed the neuronal marker, this was significantly higher than the control and mock infection groups. NT-3 transduced hfNSCs also displayed longer protruding neurites compared with other groups. Combined these results demonstrate that NT-3 modification promote the survival/proliferation, neuronal differentiation and growth of neurites of hfNSCs in vitro. This study proposes recombinant retrovirus mediated NT-3 modification may provide a promising means to resolve the poor survival and insufficient neuronal differentiation of NSCs.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Brain / embryology
  • Brain / metabolism
  • Brain / physiology
  • Cell Proliferation*
  • Cell Survival / genetics
  • Cell Survival / physiology
  • Cells, Cultured
  • Chick Embryo
  • Fetal Stem Cells / physiology*
  • Ganglia, Spinal / physiology
  • Genetic Vectors
  • Humans
  • Intermediate Filament Proteins / metabolism
  • Nerve Tissue Proteins / metabolism
  • Nestin
  • Neurites / physiology
  • Neurogenesis / genetics
  • Neurogenesis / physiology*
  • Neurons / metabolism
  • Neurons / physiology*
  • Neurotrophin 3 / genetics
  • Neurotrophin 3 / metabolism*
  • Retroviridae / genetics
  • Time Factors
  • Transduction, Genetic

Substances

  • Intermediate Filament Proteins
  • NES protein, human
  • Nerve Tissue Proteins
  • Nestin
  • Neurotrophin 3