Convergent actions of orexin/hypocretin and CRF on dopamine neurons: Emerging players in addiction

Brain Res. 2010 Feb 16:1314:139-44. doi: 10.1016/j.brainres.2009.10.068. Epub 2009 Nov 3.

Abstract

The ventral tegmental area (VTA) is a brain region centrally involved in the development and expression of a variety of behaviors associated with drug use. Orexin/hypocretin (ox/hcrt) and corticotropin releasing factor (CRF) are both peptides released into the VTA, and an increasing number of studies show that both ox/hcrt and CRF in the VTA exert powerful modulatory effects on a variety of behaviors produced by drugs of abuse. Importantly, at a cellular and synaptic level, there is strong evidence supporting the hypothesis that these neuropeptides exert similar effects onto N-methyl-d-aspartate receptors (NMDARs) expressed in VTA neurons, suggesting a common molecular target in the actions of these peptides on this circuitry and ultimately on behaviors associated with drug use. In this review, we discuss emerging evidence that ox/hcrt and CRF directly target dopamine neurons by modulating excitatory synaptic activity, and that interaction of ox/hcrt and CRF may play a central role in addictive behaviors. Understanding these interactions in more detail may suggest new therapeutic targets in the treatment of relapse to substance abuse.

Publication types

  • Research Support, N.I.H., Extramural
  • Research Support, Non-U.S. Gov't
  • Review

MeSH terms

  • Afferent Pathways / drug effects
  • Afferent Pathways / metabolism*
  • Afferent Pathways / physiopathology
  • Animals
  • Corticotropin-Releasing Hormone / metabolism*
  • Glutamic Acid / metabolism
  • Humans
  • Intracellular Signaling Peptides and Proteins / metabolism*
  • Neurons / drug effects
  • Neurons / metabolism*
  • Neuropeptides / metabolism*
  • Orexins
  • Receptors, N-Methyl-D-Aspartate / drug effects
  • Receptors, N-Methyl-D-Aspartate / metabolism
  • Substance-Related Disorders / metabolism*
  • Substance-Related Disorders / physiopathology
  • Synaptic Transmission / drug effects
  • Synaptic Transmission / physiology
  • Ventral Tegmental Area / drug effects
  • Ventral Tegmental Area / metabolism*
  • Ventral Tegmental Area / physiopathology

Substances

  • Intracellular Signaling Peptides and Proteins
  • Neuropeptides
  • Orexins
  • Receptors, N-Methyl-D-Aspartate
  • Glutamic Acid
  • Corticotropin-Releasing Hormone