Signaling pathways involved in NCAM-induced neurite outgrowth

Adv Exp Med Biol. 2010:663:151-68. doi: 10.1007/978-1-4419-1170-4_10.
No abstract available

Publication types

  • Review

MeSH terms

  • Animals
  • Calcium / metabolism
  • Calcium-Calmodulin-Dependent Protein Kinase Type 2 / metabolism
  • Cyclic AMP / metabolism
  • Cyclic AMP Response Element-Binding Protein / metabolism
  • Cyclic AMP-Dependent Protein Kinases / metabolism
  • Cyclic GMP / metabolism
  • Cytoskeletal Proteins / genetics
  • Cytoskeletal Proteins / metabolism
  • Focal Adhesion Kinase 2 / genetics
  • Focal Adhesion Kinase 2 / metabolism
  • Humans
  • Membrane Microdomains / chemistry
  • Membrane Microdomains / metabolism
  • Mitogen-Activated Protein Kinases / genetics
  • Mitogen-Activated Protein Kinases / metabolism
  • Neural Cell Adhesion Molecules / genetics
  • Neural Cell Adhesion Molecules / metabolism*
  • Neurites / physiology*
  • Phosphatidylinositol 3-Kinases / metabolism
  • Protein Isoforms / genetics
  • Protein Isoforms / metabolism*
  • Protein Kinase C / metabolism
  • Proto-Oncogene Proteins c-akt / metabolism
  • Proto-Oncogene Proteins c-fyn / genetics
  • Proto-Oncogene Proteins c-fyn / metabolism
  • Receptors, Fibroblast Growth Factor / genetics
  • Receptors, Fibroblast Growth Factor / metabolism
  • Signal Transduction / physiology*

Substances

  • Cyclic AMP Response Element-Binding Protein
  • Cytoskeletal Proteins
  • Neural Cell Adhesion Molecules
  • Protein Isoforms
  • Receptors, Fibroblast Growth Factor
  • Cyclic AMP
  • Phosphatidylinositol 3-Kinases
  • Focal Adhesion Kinase 2
  • Proto-Oncogene Proteins c-fyn
  • Proto-Oncogene Proteins c-akt
  • Cyclic AMP-Dependent Protein Kinases
  • Protein Kinase C
  • Calcium-Calmodulin-Dependent Protein Kinase Type 2
  • Mitogen-Activated Protein Kinases
  • Cyclic GMP
  • Calcium