Abstract
Quinolone and β-lactam resistance mechanisms and clonal relationships were characterized among Escherichia coli isolates resistant to ciprofloxacin and extended-spectrum cephalosporins associated with human extra-intestinal infections in Rome. The E. coli. ST131 clone was found to be prevalent. This clone invariably carried a specific pattern of substitutions in the topoisomerase genes and all isolates but one produced CTX-M-15. One ST131 isolate produced SHV-12. The new ST131 variant described here is of particular concern because it combines fluoroquinolone resistance and chromosomally encoded CTX-M-15.
© 2010 The Authors. Journal Compilation © 2010 European Society of Clinical Microbiology and Infectious Diseases.
Publication types
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Research Support, Non-U.S. Gov't
MeSH terms
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Amino Acid Substitution / genetics
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Anti-Bacterial Agents / pharmacology
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Bacterial Typing Techniques
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Ciprofloxacin / pharmacology*
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Cluster Analysis
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DNA Topoisomerase IV / genetics
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Drug Resistance, Multiple, Bacterial*
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Escherichia coli / classification
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Escherichia coli / drug effects*
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Escherichia coli / enzymology*
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Escherichia coli / isolation & purification
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Escherichia coli Infections / epidemiology*
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Escherichia coli Infections / microbiology
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Humans
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Microbial Sensitivity Tests
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Molecular Epidemiology
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Molecular Typing
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Mutation, Missense
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Rome / epidemiology
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beta-Lactamases / biosynthesis*
Substances
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Anti-Bacterial Agents
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Ciprofloxacin
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beta-lactamase CTX-M-15
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beta-Lactamases
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DNA Topoisomerase IV