Abstract
To assess the effect of potential therapeutic agents in dystrophic mice it is useful to have a functional test regime that does not affect the natural disease progression of mdx mice with dystrophinopathy. We determined the effect of a 12 week test regime consisting of fore limb grip strength, rotarod analysis and two and four limb hanging wire tests on the disease progression of 4-week-old mdx mice. Mice performed the different functional tests on consecutive days on a weekly basis. No difference was found in serum creatine kinase levels between functionally active and sedentary mice. The percentage of fibrotic/necrotic areas assessed in a semi-automated way with colour deconvolution of skeletal muscles, heart and diaphragm did not vary within muscles or between groups, nor did the gene expression levels of disease-related genes. We conclude that this test regime may be suitable for short-term functional evaluation of therapeutic approaches in the mdx mouse.
Copyright 2010 Elsevier B.V. All rights reserved.
Publication types
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Research Support, Non-U.S. Gov't
MeSH terms
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Age Factors
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Animals
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Creatine Kinase / analysis
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Creatine Kinase / blood
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Disability Evaluation*
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Disease Models, Animal
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Fibrosis / drug therapy
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Fibrosis / metabolism
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Fibrosis / physiopathology
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Gene Expression Regulation / genetics
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Heart / drug effects
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Heart / physiopathology
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Image Processing, Computer-Assisted
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Male
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Mice
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Mice, Inbred mdx
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Motor Activity / drug effects
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Motor Activity / genetics
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Muscle Fibers, Skeletal / metabolism
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Muscle Fibers, Skeletal / pathology
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Muscle Proteins / genetics
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Muscle Strength / drug effects
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Muscle Strength / genetics
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Muscle, Skeletal / drug effects
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Muscle, Skeletal / metabolism
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Muscle, Skeletal / physiopathology
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Muscular Dystrophy, Duchenne / diagnosis*
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Muscular Dystrophy, Duchenne / drug therapy
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Muscular Dystrophy, Duchenne / physiopathology*
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Myocardium / metabolism
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Neurologic Examination / methods
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Outcome Assessment, Health Care / methods*
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Predictive Value of Tests
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Recovery of Function / drug effects
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Recovery of Function / genetics
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Severity of Illness Index*
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Time Factors
Substances
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Muscle Proteins
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Creatine Kinase