[Cyclic strain promotes migration of human periodontal ligament cell via extracellular signal-regulated kinase (ERK) signaling pathway]

Zhonghua Kou Qiang Yi Xue Za Zhi. 2010 Feb;45(2):80-4.
[Article in Chinese]

Abstract

Objective: To study the effect of cyclic strain on migration of human periodontal ligament cell(hPDLC) and underlying mechanism.

Methods: The cultured hPDLC were subjected to 10% or 20%-elongation magnitude cyclic strain at frequency of 0.1 Hz by FX-4000T system for 6 or 24 hours-duration respectively, while the static group serves as control. hPDLC migration was assayed by wound healing method. The expressions of matrix metalloproteinases-9 (MMP-9) and p-ERK1/2 in hPDLC without or with cyclic strain were analyzed by Western blotting. To investigate the effect of ERK signaling pathway and MMP-9 on migration of hPDLC, the cells were incubated with PD98059, a specific extracellular signal-regulated kinase (ERK) kinase inhibitor, or doxycycline, a MMP inhibitor. Then the expressions of p-ERK1/2 and MMP-9 and hPDLC migration were analyzed.

Results: In wound healing tests, the migration of hPDLC exposed to 10% or 20%-cyclic strain at 0.1 Hz-frequency for 6 hours was not apparent but became significantly different for 24 hours (P < 0.05) compared to control. Furthermore, the 20%-elongation magnitude of cyclic strain had more remarkable effect on migration of hPDLC than 10%-elongation magnitude at 24 hours-duration (P < 0.05). Cyclic strain obviously increased the expression of MMP-9 in hPDLC (P < 0.05). PD98059 could repress not only the activation of p-ERK1/2 but also the expression of MMP-9 induced by cyclic strain in hPDLC. The migration of hPDLC enhanced by cyclic strain was repressed by DOX or PD98059 in wound healing tests.

Conclusions: Cyclic strain promotes the migration of hPDLC through activating ERK signaling pathway and inducing the expression of MMP-9.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adolescent
  • Cell Movement* / drug effects
  • Cells, Cultured
  • Doxycycline / pharmacology
  • Flavonoids / pharmacology
  • Humans
  • MAP Kinase Signaling System / drug effects*
  • Male
  • Matrix Metalloproteinase 9 / metabolism*
  • Matrix Metalloproteinase Inhibitors / pharmacology
  • Periodontal Ligament / cytology*
  • Periodontal Ligament / metabolism
  • Protein Kinase Inhibitors / pharmacology
  • Stress, Mechanical*

Substances

  • Flavonoids
  • Matrix Metalloproteinase Inhibitors
  • Protein Kinase Inhibitors
  • MMP9 protein, human
  • Matrix Metalloproteinase 9
  • Doxycycline
  • 2-(2-amino-3-methoxyphenyl)-4H-1-benzopyran-4-one