Hypoxia, HIFs and bone development

Bone. 2010 Aug;47(2):190-6. doi: 10.1016/j.bone.2010.04.606. Epub 2010 May 2.

Abstract

Oxygen is not only an obviously important substrate, but it is also a regulatory signal that controls expression of a specific genetic program. Crucial mediator of the adaptive response of cells to hypoxia is the family of Hypoxia-Inducible Transcription Factors (HIFs).The fetal growth plate, which is an avascular structure of mesenchymal origin, has a unique out-in gradient of oxygenation. HIF-1alpha is necessary for chondrogenesis in vivo by controlling a complex homeostatic response that allows chondrocytes to survive and differentiate in a hypoxic environment. Moreover, HIFs are also essential in osteogenesis and joint development. This brief Perspective summarizes the critical role of HIFs in endochondral bone development.

Publication types

  • Research Support, N.I.H., Extramural

MeSH terms

  • Animals
  • Bone Development / physiology*
  • Cell Hypoxia
  • Chondrogenesis / physiology
  • Humans
  • Hypoxia-Inducible Factor 1 / metabolism*
  • Joints / metabolism
  • Models, Biological

Substances

  • Hypoxia-Inducible Factor 1