Roles of the metabolic syndrome, HDL cholesterol, and coronary atherosclerosis in subclinical inflammation

Diabetes Care. 2010 Aug;33(8):1853-5. doi: 10.2337/dc09-2376. Epub 2010 May 18.

Abstract

Objective: The metabolic syndrome (MetS) and coronary artery disease (CAD) frequently coincide; their individual contribution to inflammation is unknown.

Research design and methods: We enrolled 1,010 patients undergoing coronary angiography. Coronary stenoses >or=50% were considered significant. The MetS was defined according to American Heart Association-revised National Cholesterol Education Program Adult Treatment Panel III criteria.

Results: C-reactive protein (CRP) did not differ between patients with significant CAD and subjects without significant CAD (P = 0.706) but was significantly higher in MetS patients than in those without MetS (P < 0.001). The MetS criteria low HDL cholesterol (P < 0.001), large waist (P < 0.001), high glucose (P < 0.001), and high blood pressure (P = 0.016), but not high triglycerides (P = 0.352), proved associated with CRP. When all MetS traits were considered simultaneously, only low HDL cholesterol proved independently associated with CRP (F = 44.19; P < 0.001).

Conclusions: CRP is strongly associated with the MetS but not with coronary atherosclerosis. The association of the MetS with subclinical inflammation is driven by the low HDL cholesterol feature.

MeSH terms

  • C-Reactive Protein / metabolism
  • Cholesterol, HDL / metabolism*
  • Coronary Angiography
  • Coronary Artery Disease / metabolism*
  • Humans
  • Metabolic Syndrome / metabolism*
  • Risk Factors

Substances

  • Cholesterol, HDL
  • C-Reactive Protein