The transcriptional repression of platelet-derived growth factor receptor-beta by the zinc finger transcription factor ZNF24

Biochem Biophys Res Commun. 2010 Jun 25;397(2):318-22. doi: 10.1016/j.bbrc.2010.05.110. Epub 2010 May 26.

Abstract

The zinc finger transcription factor ZNF24 is a relatively uncharacterized Krüppel-like transcription factor, displaying a suppressive effect on the transcription. ZNF24 is involved in negative regulation of vascular endothelial growth factor (VEGF) and may represent a novel repressor of VEGF transcription. In a previous study, our analysis reveals that ZNF24 is a pleiotropic factor that has a role in hematopoiesis, brain development and cancers. Activation of PDGFR-beta is critically involved into various cell functions including recruitment of stromal cells and VEGF induction in tumor and perivascular cells. Here, overexpression of ZNF24 resulted in a significant down-regulation of PDGFR-beta, whereas silencing of ZNF24 with small interfering RNA led to increased PDGFR-beta expression. Overexpression of ZNF24 resulted in a significant decrease in PDGFR-beta promoter activity. These data suggest that ZNF24 is involved in negative regulation of PDGFR-beta and may represent a novel repressor of PDGFR-beta transcription.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Cell Line
  • Cell Line, Tumor
  • Down-Regulation
  • Gene Expression Regulation*
  • Gene Knockdown Techniques
  • Humans
  • Kruppel-Like Transcription Factors / genetics
  • Kruppel-Like Transcription Factors / metabolism*
  • Promoter Regions, Genetic
  • RNA, Small Interfering / genetics
  • Receptor, Platelet-Derived Growth Factor beta / genetics*
  • Repressor Proteins / genetics
  • Repressor Proteins / metabolism*
  • Transcription, Genetic
  • Up-Regulation
  • Zinc Fingers*

Substances

  • Kruppel-Like Transcription Factors
  • RNA, Small Interfering
  • Repressor Proteins
  • ZNF24 protein, human
  • Receptor, Platelet-Derived Growth Factor beta