[Higher percentages of T regulatory cells in children at risk for developing type 1 diabetes mellitus]

Pediatr Endocrinol Diabetes Metab. 2010;16(1):7-10.
[Article in Polish]

Abstract

Introduction: The natural history of type 1 diabetes is concerned with the appearance of autoantibodies against antigens of pancreatic beta cells. The last decade revealed some evidence of the participation of T regulatory lymphocytes - cells which suppress immune response - in the pathogenesis of type 1 diabetes and prediabetes.

Aim of the study: was the assessment of T regulatory cells in the blood of children at risk for developing type 1 the diabetes mellitus.

Material and methods: 85 subjects, siblings of children with type 1 diabetes, were enrolled into the study. The presence of anti-GAD65 antibodies was assessed. With the use of flow cytometry the following cell subpopulations were noted: CD4+, CD4+CD25high and CD4+CD25highCD127low with the coexpression of: CD28, CD45RO, CD54, CD62L and CD134 molecules.

Results: We did not observe any differences in white blood cell count, lymphocyte (including CD4+) count and the percentage between the examined and control groups. We noted higher percentages of T regulatory cells: CD4+CD25high, CD4+CD127low and CD4+CD25highCD127low in children with the presence of anti-GAD65 antibodies as compared to the control children.

Conclusion: Higher percentages of T regulatory cells in the blood of children with the presence of anti-GAD65 antibodies may suggest an intensive regulatory response present in patients at risk for developing type 1 diabetes.

Publication types

  • English Abstract

MeSH terms

  • Antibodies, Anti-Idiotypic / blood*
  • Antigens, CD / metabolism
  • Child
  • Diabetes Mellitus, Type 1 / immunology*
  • Female
  • Flow Cytometry
  • Glutamate Decarboxylase / immunology*
  • Humans
  • Lymphocyte Count
  • Male
  • T-Lymphocytes, Regulatory / immunology*

Substances

  • Antibodies, Anti-Idiotypic
  • Antigens, CD
  • Glutamate Decarboxylase
  • glutamate decarboxylase 2