Abstract
The most common genetic abnormalities of ependymomas involve the chromosome 22 where there is the oncosuppressor gene neurofibromin 2 (NF2). NF2 mutations are primarily encountered in spinal lesions. In contrast, NF2 alterations do not seem related to tumor grade. We studied the NF2 expression through a real-time polymerase chain reaction in 25 pediatric anaplastic ependymomas. We compared the NF2 expression in neoplastic and non-neoplastic tissues, in supratentorial and infratentorial ependymomas and in primitive and non-primitive tumors (recurrences and metastases). Statistical analysis did not prove significant differences. Our results suggest that NF2 alterations are not typical of intracranial anaplastic ependymomas.
MeSH terms
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Adolescent
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Brain Neoplasms / genetics*
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Brain Neoplasms / pathology
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Brain Neoplasms / surgery
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Child
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Child, Preschool
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DNA, Neoplasm / analysis
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Ependymoma / genetics*
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Ependymoma / secondary
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Ependymoma / surgery
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Female
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Gene Expression
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Humans
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Infant
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Infratentorial Neoplasms / genetics
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Infratentorial Neoplasms / pathology
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Infratentorial Neoplasms / surgery
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Male
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Neoplasm Metastasis
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Neoplasm Recurrence, Local / genetics
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Neoplasm Recurrence, Local / pathology
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Neurofibromin 2 / genetics*
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RNA, Messenger / metabolism
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Reverse Transcriptase Polymerase Chain Reaction / methods*
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Supratentorial Neoplasms / genetics
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Supratentorial Neoplasms / pathology
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Supratentorial Neoplasms / surgery
Substances
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DNA, Neoplasm
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Neurofibromin 2
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RNA, Messenger