Genomic predictors of prostate cancer therapy outcomes

Expert Rev Mol Diagn. 2010 Jul;10(5):619-36. doi: 10.1586/erm.10.53.

Abstract

Although numerous attempts have been made to forecast outcomes for prostate cancer after therapy using clinical and histological variables, the ability to accurately predict an individual's response to a specific treatment remains elusive. Recently, major advances in the field of genomics have made possible the near-comprehensive assessment of the genetic status of tumor genomes, with major concentration on predicting an individual's response to a specific treatment. Genomic approaches to treatment response include, but are not limited to, detection of gene rearrangements, DNA copy-number aberrations, single-nucleotide polymorphisms, epigenetic changes and differential gene-expression patterns. These approaches have been used to predict response to treatment for local and systemic disease in multiple small cohorts. Further study with larger cohorts and longer follow-up should result in more concordance among genomic approaches, and will enable physicians to gain insight into the heterogeneity of supposedly 'similar' cancers and help tailor treatments accordingly.

MeSH terms

  • Animals
  • Epigenesis, Genetic
  • Gene Expression Profiling
  • Genome, Human
  • Genomics* / methods
  • Humans
  • Male
  • MicroRNAs / genetics
  • MicroRNAs / metabolism
  • Mutation
  • Oncogene Proteins, Fusion / genetics
  • PTEN Phosphohydrolase / genetics
  • Prostatic Neoplasms / diagnosis
  • Prostatic Neoplasms / genetics*
  • Prostatic Neoplasms / therapy*
  • Treatment Outcome*

Substances

  • MicroRNAs
  • Oncogene Proteins, Fusion
  • TMPRSS2-ETS fusion protein, human
  • PTEN Phosphohydrolase
  • PTEN protein, human