The role of 9-O-acetylated ganglioside D3 (CD60) and {alpha}4{beta}1 (CD49d) expression in predicting the survival of patients with Sezary syndrome

Haematologica. 2010 Nov;95(11):1905-12. doi: 10.3324/haematol.2010.026260. Epub 2010 Jul 27.

Abstract

Background: Sézary syndrome is a rare and very aggressive leukemic variant of cutaneous T-cell lymphoma characterized by extensive skin involvement and a malignant circulating CD4(+) T-cell clone which homes to the skin, over-expresses CD60, and lacks CD7, CD26 and CD49d. So far prognostic markers in this disease are limited to treatment with systemic steroids, age, serum lactate dehydrogenase, and a white blood cell count of 20×10(9)/L or higher: no other biological marker with prognostic value, especially related to malignant cells, has been described.

Design and methods: We used flow activated cell sorting analysis to compare the distribution of the T-cell receptor-Vβ repertoire and several surface molecules (CD7, CD26, CD49d and CD60) within the circulating CD4(+) T-cell population in 62 patients with Sézary syndrome, 180 with mycosis fungoides, 6 with B-cell lymphomas, and 19 with chronic eczema. We calculated the 5-year overall survival of patients with Sézary syndrome after first hospital admission using Kaplan-Meier product-limit estimates and hazard ratios from the Cox proportional hazards model.

Results: We found that both higher number of CD60(+) and lower number of CD49d(+) cells within circulating CD4(+) T cells at disease presentation were significantly associated with a lower probability of survival. An exceedingly high risk of death was observed for patients with a combination of a high proportion of CD4(+)CD60(+) cells (≥ 0.5×10(9)/L) and low proportion of CD4(+)CD49d(+) cells (<0.5×10(9)/L) (hazard ratio = 12.303, 95% confidence interval 1.5-95.9; P<0.02). In addition, a skewed usage of T-cell receptor-Vβ subfamilies was observed in the circulating T-cell clone for 61.9% of all patients with Sézary syndrome, T-cell receptor-Vβ 2 and 5.1 subfamilies being the most frequently represented (42.8%), followed by T-cell receptor-Vβ 12 and 13.1.

Conclusions: In this study we showed that up-regulation of CD60 and down-regulation of CD49d on circulating CD4(+) T cells are two useful markers for predicting a very poor outcome in patients with Sézary syndrome.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adult
  • Aged
  • Antigens, CD7 / blood
  • Biomarkers, Tumor / blood*
  • CD4-Positive T-Lymphocytes / metabolism*
  • Dipeptidyl Peptidase 4 / blood
  • Disease-Free Survival
  • Female
  • Flow Cytometry
  • Gangliosides / blood*
  • Gene Expression Regulation, Neoplastic
  • Humans
  • Integrin alpha4 / blood*
  • Male
  • Middle Aged
  • Receptors, Antigen, T-Cell, alpha-beta / metabolism
  • Sezary Syndrome* / blood
  • Sezary Syndrome* / mortality
  • Skin Neoplasms* / blood
  • Skin Neoplasms* / mortality
  • Survival Rate

Substances

  • Antigens, CD7
  • Biomarkers, Tumor
  • Gangliosides
  • Receptors, Antigen, T-Cell, alpha-beta
  • Integrin alpha4
  • 9-O-acetyl-GD3 ganglioside
  • DPP4 protein, human
  • Dipeptidyl Peptidase 4