A role for ATM kinase activity and Mre11 in microhomology-mediated end-joining

Cell Cycle. 2010 Aug 15;9(16):3147-8. doi: 10.4161/cc.9.16.12814. Epub 2010 Aug 15.

Authors

Anthony J Davis¹, David J Chen

Affiliation

¹ University of Texas Southwestern Medical Center, Dallas, TX, USA.

PMID: 20814230
DOI: 10.4161/cc.9.16.12814

No abstract available

Publication types

News
Comment

MeSH terms

Ataxia Telangiectasia Mutated Proteins
Cell Cycle Proteins / metabolism*
DNA Breaks, Double-Stranded
DNA Repair*
DNA-Binding Proteins / antagonists & inhibitors
DNA-Binding Proteins / genetics
DNA-Binding Proteins / metabolism*
Humans
MRE11 Homologue Protein
Protein Serine-Threonine Kinases / metabolism*
RNA Interference
Tumor Suppressor Proteins / metabolism*

Substances

Cell Cycle Proteins
DNA-Binding Proteins
MRE11 protein, human
Tumor Suppressor Proteins
ATM protein, human
Ataxia Telangiectasia Mutated Proteins
Protein Serine-Threonine Kinases
MRE11 Homologue Protein