Effects of cardiac myosin binding protein-C on the regulation of interaction of cardiac myosin with thin filament in an in vitro motility assay

Biochem Biophys Res Commun. 2010 Oct 8;401(1):159-63. doi: 10.1016/j.bbrc.2010.09.040. Epub 2010 Sep 16.

Abstract

Modulatory role of whole cardiac myosin binding protein-C (сMyBP-C) in regulation of cardiac muscle contractility was studied in the in vitro motility assay with rabbit cardiac myosin as a motor protein. The effects of cMyBP-C on the interaction of cardiac myosin with regulated thin filament were tested in both in vitro motility and ATPase assays. We demonstrate that the addition of cMyBP-C increases calcium regulated Mg-ATPase activity of cardiac myosin at submaximal calcium. The Hill coefficient for 'pCa-velocity' relation in the in vitro motility assay decreased and the calcium sensitivity increased when сMyBP-C was added. Results of our experiments testifies in favor of the hypothesis that сMyBP-C slows down cross-bridge kinetics when binding to actin.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Actin Cytoskeleton / metabolism*
  • Adenosine Triphosphatases / metabolism
  • Animals
  • Biological Assay
  • Calcium / metabolism
  • Cardiac Myosins / metabolism*
  • Carrier Proteins / metabolism*
  • Magnesium / metabolism
  • Myocardial Contraction*
  • Rabbits

Substances

  • Carrier Proteins
  • myosin-binding protein C
  • Adenosine Triphosphatases
  • Cardiac Myosins
  • Magnesium
  • Calcium