Abstract
Starting from the initial bis-anilinopyrimidine 1, good potency against EphB4 was retained when benzodioxole at C-4 was replaced by an indazole. The key interactions of the indazole with the protein were characterised by crystallographic studies. Further optimisation led to compound 20, a potent inhibitor of the EphB4 and Src kinases with good pharmacokinetics in various preclinical species and high fraction unbound in plasma. Compound 20 may be used as a tool for evaluating the potential of EphB4 kinase inhibitors in vivo.
Copyright © 2010 Elsevier Ltd. All rights reserved.
MeSH terms
-
Animals
-
Benzodioxoles / chemical synthesis*
-
Benzodioxoles / pharmacokinetics
-
Benzodioxoles / pharmacology*
-
CHO Cells
-
Cricetinae
-
Cricetulus
-
Crystallography, X-Ray
-
Dogs
-
Female
-
Male
-
Mice
-
Mice, Nude
-
Models, Molecular
-
Phosphorylation
-
Protein Kinase Inhibitors / chemical synthesis*
-
Protein Kinase Inhibitors / pharmacokinetics
-
Protein Kinase Inhibitors / pharmacology*
-
Protein-Tyrosine Kinases / antagonists & inhibitors*
-
Rats
-
Rats, Wistar
-
Receptor, EphB4 / antagonists & inhibitors*
-
Structure-Activity Relationship
-
Substrate Specificity
-
src-Family Kinases / antagonists & inhibitors
Substances
-
Benzodioxoles
-
Protein Kinase Inhibitors
-
Protein-Tyrosine Kinases
-
Receptor, EphB4
-
src-Family Kinases