Role of metabolism in 1-bromopropane-induced hepatotoxicity in mice

J Toxicol Environ Health A. 2010;73(21-22):1431-40. doi: 10.1080/15287394.2010.511546.

Abstract

A possible role of metabolism in 1-bromopropane (1-BP)-induced hepatotoxicity was investigated in male ICR mice. The depletion of glutathione (GSH) by formation of GSH conjugates was associated with increased hepatotoxicity in 1-BP-treated mice. The formation of S-propyl and 2-hydroxypropyl GSH conjugates were identified in the liver following 1-BP treatment. In addition, the formation of reactive metabolites of 1-BP by certain cytochrome P-450 (CYP) may be involved in 1-BP-induced hepatotoxicity. The decreased content of hepatic GSH produced by 1-BP was associated not only with increased activities of alanine aminotransferase (ALT) and aspartate aminotransferase (AST) but also with elevated levels of hepatic thiobarbituric acid-reactive substance (TBARS) in mice where metabolic enzymes were induced by pretreatment with phenobarbital. In addition, the hepatotoxicity induced by 1-BP was prevented by pretreatment with SKF-525A. Taken together, the formation of reactive metabolites by CYP and depletion of GSH may play important roles in hepatotoxicity induced by 1-BP.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Alanine Transaminase / blood
  • Animals
  • Aspartate Aminotransferases / blood
  • Chemical and Drug Induced Liver Injury / etiology
  • Chemical and Drug Induced Liver Injury / metabolism*
  • Chromatography, High Pressure Liquid
  • Cytochrome P-450 Enzyme Inhibitors
  • Cytochrome P-450 Enzyme System / metabolism
  • Enzyme Induction
  • Enzyme Inhibitors / pharmacology
  • Glutathione / analogs & derivatives
  • Glutathione / drug effects
  • Glutathione / metabolism
  • Hydrocarbons, Brominated / metabolism
  • Hydrocarbons, Brominated / toxicity
  • Liver / drug effects
  • Liver / metabolism*
  • Male
  • Mice
  • Mice, Inbred ICR
  • Phenobarbital / pharmacology
  • Proadifen / pharmacology
  • Specific Pathogen-Free Organisms
  • Spectrometry, Mass, Electrospray Ionization
  • Thiobarbituric Acid Reactive Substances / metabolism

Substances

  • Cytochrome P-450 Enzyme Inhibitors
  • Enzyme Inhibitors
  • Hydrocarbons, Brominated
  • Thiobarbituric Acid Reactive Substances
  • Cytochrome P-450 Enzyme System
  • Proadifen
  • Aspartate Aminotransferases
  • Alanine Transaminase
  • Glutathione
  • 1-bromopropane
  • Phenobarbital