Variability in G-protein-coupled signaling studied with microfluidic devices

Biophys J. 2010 Oct 20;99(8):2414-22. doi: 10.1016/j.bpj.2010.08.043.

Abstract

Different cells, even those that are genetically identical, can respond differently to identical stimuli, but the precise source of this variability remains obscure. To study this problem, we built a microfluidic experimental system which can track responses of individual cells across multiple stimulations. We used this system to determine that amplitude variation in G-protein-activated calcium release in RAW264.7 macrophages is generally extrinsic, i.e., they arise from long-lived variations between cells and not from stochastic activation of signaling components. In the case of responses linked to P2Y family purine receptors, we estimate that approximately one-third of the observed variability in calcium release is receptor-specific. We further demonstrate that the signaling apparatus downstream of P2Y6 receptor activation is moderately saturable. These observations will be useful in constructing and constraining single-cell models of G protein-coupled calcium dynamics.

Publication types

  • Research Support, N.I.H., Extramural
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Calcium Signaling / drug effects
  • Cell Culture Techniques
  • Cell Line
  • Cytological Techniques / instrumentation*
  • Injections
  • Kinetics
  • Macrophages / cytology
  • Macrophages / metabolism
  • Mice
  • Microfluidic Analytical Techniques*
  • Receptors, G-Protein-Coupled / metabolism*
  • Signal Transduction* / drug effects
  • Uridine Diphosphate / pharmacology

Substances

  • Receptors, G-Protein-Coupled
  • Uridine Diphosphate