Suppressive effects of sivelestat on interleukin 8 and TNF-α production from LPS-stimulated granulocytes in whole blood culture

J Anesth. 2010 Dec;24(6):901-7. doi: 10.1007/s00540-010-1030-2. Epub 2010 Oct 26.

Abstract

Purpose: The goal of the study was to examine the effects of sivelestat sodium hydrate (sivelestat), a neutrophil elastase inhibitor, on production of cytokines in granulocytes and monocytes, using flow cytometry after cytokine staining in whole blood culture.

Methods: Blood samples were collected from healthy volunteers. Vehicle (control group), lipopolysaccharide (LPS) (LPS group), or LPS + sivelestat (sivelestat group) were added to the whole blood, followed by addition of a protein transport inhibitor in each group. After incubation, staining for cytokines retained in the cells was performed by addition of an anti-interleukin 8 (IL-8) or anti-tumor necrosis factor-α (TNF-α) antibody. The cells were then analyzed using flow cytometry.

Results: Granulocytic production of IL-8 induced by 1 ng/ml LPS was significantly (P < 0.05) inhibited by treatment with 1 μg/ml sivelestat, and upregulation of IL-8 by 10 ng/ml LPS was also significantly (P < 0.05) suppressed by 1 and 10 μg/ml sivelestat. Addition of 10 or 100 μg/ml sivelestat significantly (P < 0.05) inhibited the production of TNF-α from granulocytes induced by 10 ng/ml LPS. Sivelestat did not significantly inhibit LPS-induced monocytic production of TNF-α and IL-8.

Conclusion: Suppression of granulocytic production of IL-8 and TNF-α by sivelestat suggests that this drug may be useful for treatment of morbid conditions involving IL-8 and TNF-α at onset.

MeSH terms

  • Acute Lung Injury / drug therapy
  • Acute Lung Injury / physiopathology
  • Flow Cytometry
  • Glycine / analogs & derivatives*
  • Glycine / pharmacology
  • Granulocytes / drug effects
  • Granulocytes / metabolism*
  • Humans
  • In Vitro Techniques
  • Interleukin-8 / biosynthesis*
  • Lipopolysaccharides / pharmacology*
  • Monocytes / drug effects
  • Monocytes / metabolism
  • Proteinase Inhibitory Proteins, Secretory / pharmacology*
  • Sulfonamides / pharmacology*
  • Tumor Necrosis Factor-alpha / biosynthesis*

Substances

  • Interleukin-8
  • Lipopolysaccharides
  • Proteinase Inhibitory Proteins, Secretory
  • Sulfonamides
  • Tumor Necrosis Factor-alpha
  • sivelestat
  • Glycine