Aortic endothelial cell damage induced by beta-VLDL and macrophages in vitro

Atherosclerosis. 1990 Dec;85(2-3):161-7. doi: 10.1016/0021-9150(90)90107-t.

Abstract

We performed an in vitro study to assess damage to swine aortic endothelial cells by rabbit beta-VLDL and/or rabbit peritoneal macrophages. Incubation of cultured aortic endothelial cells with beta-VLDL, macrophages, or macrophage lysate induced endothelial cell damage time- and dose-dependently as estimated by [3H]adenine release. Incubation of endothelial cells with both beta-VLDL and macrophages produced a synergistic effect on the increase of [3H]adenine release. Pretreatment of the endothelial cells with some kinds of antioxidants (probucol 50 micrograms/ml, vitamin E 50 microM, superoxide dismutase-polyethylene glycol 0.5 mg/ml, or catalase-polyethylene glycol 0.5-1.0 mg/ml) significantly prevented the endothelial damage by beta-VLDL or macrophage lysate. We conclude that beta-VLDL and/or macrophages could induce endothelial cell damage and that some kinds of antioxidants could prevent it.

MeSH terms

  • Adenine / metabolism
  • Animals
  • Antioxidants / pharmacology
  • Aorta, Thoracic / metabolism
  • Aorta, Thoracic / pathology
  • Catalase / pharmacology
  • Cells, Cultured
  • Endothelium, Vascular / drug effects
  • Endothelium, Vascular / metabolism
  • Endothelium, Vascular / pathology*
  • Lipoproteins, VLDL / physiology*
  • Lipoproteins, VLDL / toxicity
  • Macrophages / physiology*
  • Male
  • Polyethylene Glycols / pharmacology
  • Probucol / pharmacology
  • Rabbits
  • Superoxide Dismutase / pharmacology
  • Swine
  • Vitamin E / pharmacology

Substances

  • Antioxidants
  • Lipoproteins, VLDL
  • catalase-polyethylene glycol
  • Vitamin E
  • Polyethylene Glycols
  • Catalase
  • Superoxide Dismutase
  • polyethylene glycol-superoxide dismutase
  • Adenine
  • Probucol