Lipopolysaccharide-unresponsive mutant pre-B-cell lines blocked in NF-kappa B activation

Mol Cell Biol. 1990 Jan;10(1):422-5. doi: 10.1128/mcb.10.1.422-425.1990.

Abstract

NF-kappa B activation is a crucial late step in the induction of immunoglobulin kappa light-chain gene expression in pre-B cells by lipopolysaccharide (LPS). We have analyzed NF-kappa B activation in three independent mutant lines of 70Z/3 pre-B cells which are unresponsive to LPS. All three variant cell lines failed to activate NF-kappa B when induced with LPS or the phorbol ester 12-O-tetradecanoylphorbol 13-acetate. However, all three cell lines contained functional NF-kappa B, as revealed by detergent treatment of cytoplasmic extracts. Moreover, cycloheximide induced limited activation of NF-kappa B comparable to that in wild-type 70Z/3 pre-B cells in two of the three variant lines. These results indicate that the mutations blocking kappa gene induction in these variant 70Z/3 pre-B-cell lines affect NF-kappa B activation.

Publication types

  • Research Support, Non-U.S. Gov't
  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Animals
  • B-Lymphocytes / physiology*
  • Gene Expression Regulation / drug effects
  • Gene Rearrangement
  • Genes, Immunoglobulin
  • Immunoglobulin kappa-Chains / genetics*
  • In Vitro Techniques
  • Lipopolysaccharides / pharmacology*
  • Lymphocyte Activation
  • Mice
  • Mutation
  • NF-kappa B
  • Tetradecanoylphorbol Acetate / pharmacology
  • Transcription Factors / physiology*
  • Transcriptional Activation

Substances

  • Immunoglobulin kappa-Chains
  • Lipopolysaccharides
  • NF-kappa B
  • Transcription Factors
  • Tetradecanoylphorbol Acetate