IRF6 mutations in mixed isolated familial clefting

Am J Med Genet A. 2010 Dec;152A(12):3107-9. doi: 10.1002/ajmg.a.33053.

Abstract

Mutations in the interferon regulatory factor 6 (IRF6) gene are known to cause van der Woude syndrome (VWS), a common syndromic form of oro-facial clefting characterized by the familial occurrence of mixed clefting (cleft lip with or without a cleft palate and cleft palate alone in the same family) and lower lip pits. As lip pits are not present in all cases of VWS, IRF6 mutations can cause a phenotype identical to non-syndromic clefting. However, recent studies failed to identify IRF6 mutations in sporadic and familial non-syndromic clefting, concluding that testing for IRF6 was not warranted for sporadic or familial non-syndromic clefting. Here we report on two families that demonstrate familial mixed clefting in which mutations in IRF6 were identified, suggesting that IRF6 testing does have a role in familial, non-syndromic OFC.

Publication types

  • Case Reports

MeSH terms

  • Abnormalities, Multiple / genetics
  • Child, Preschool
  • Cleft Lip / genetics*
  • Cleft Palate / genetics*
  • Family
  • Female
  • Heterozygote
  • Humans
  • Infant
  • Interferon Regulatory Factors / genetics*
  • Male
  • Mutation*
  • Mutation, Missense
  • Pedigree
  • Phenotype
  • Syndrome

Substances

  • IRF6 protein, human
  • Interferon Regulatory Factors