We present a 46-year-old man who developed repetitive focal tonic-clonic seizure-like involuntary movement in the left lower extremity after a left basal ganglia (BG) hemorrhage. His symptoms were induced only by voluntary movement of the left lower extremity and were followed by transient paralysis. Electroencephalography during an attack showed no epileptiform discharges. Carbamazepine completely abolished his symptoms. This report implies that damage to the BG and/or its connections might cause seizure-like involuntary movement.
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