[Analysis of frequency of peripheral blood CD4+; CD25(high);Tregs and CD4+; CD25(low);T cells and expression of PD-1 in SLE and RA patients]

Xi Bao Yu Fen Zi Mian Yi Xue Za Zhi. 2011 Jan;27(1):23-5.
[Article in Chinese]

Abstract

Aim: To explore the frequencies of peripheral blood CD4(+);CD25(high);Treg and CD4(+);CD25(low);T cells and the expression of the co-stimulatory molecule PD-1 on these two group cells surface in SLE and RA patients, and to explore their roles in cell immunity disorder of SLE and RA.

Methods: Flow cytometry (FCM) was used to determine the frequencies of peripheral blood CD4(+);CD25(high);Treg and CD4(+);CD25(low);T cells, and the expression percentage of PD-1.

Results: Compared with healthy control, the frequencies of peripheral blood CD4(+);CD25(high);Treg from both SLE and RA patients groups decreased significantly(P<0.05). Compared between two disease groups, the frequency of peripheral blood CD4(+);CD25(high);Treg in SLE patients was significantly lower(P<0.05). The expression percentage of PD-1 on CD4(+);CD25(high);Treg surface in RA group was obviously lower than that in both healthy control and SLE patients groups(P<0.05), while the percentage had no significant difference between SLE patients and healthy control(P>0.05). There was no significant difference in both the frequency of CD4(+);CD25(low);T cells and the expression percentage of PD-1 on this subset cells among three groups.

Conclusion: The weakened ability of peripheral blood CD4(+);CD25(high);Treg to suppress effector T cells resulted from their production deficiency is the common characteristic of SLE and RA patients. Decreased expression of PD-1 is the primary cause leading to the suppressive function of peripheral blood CD4(+);CD25(high);Treg weakened in RA patients. However, PD-1 does not play major role in weakening the suppression activity of CD4(+);CD25(high);Treg from SLE patients.

MeSH terms

  • Adolescent
  • Adult
  • Aged
  • Antigens, CD / biosynthesis*
  • Antigens, CD / blood
  • Apoptosis Regulatory Proteins / biosynthesis*
  • Apoptosis Regulatory Proteins / blood
  • Arthritis, Rheumatoid / blood
  • Arthritis, Rheumatoid / immunology*
  • CD4 Antigens / biosynthesis*
  • CD4 Antigens / blood
  • Cells, Cultured
  • Female
  • Flow Cytometry / methods
  • Humans
  • Interleukin-2 Receptor alpha Subunit / biosynthesis*
  • Interleukin-2 Receptor alpha Subunit / blood
  • Lupus Erythematosus, Systemic / blood
  • Lupus Erythematosus, Systemic / immunology*
  • Male
  • Middle Aged
  • Programmed Cell Death 1 Receptor
  • T-Lymphocytes / immunology
  • T-Lymphocytes / metabolism
  • T-Lymphocytes, Regulatory / immunology*
  • T-Lymphocytes, Regulatory / metabolism
  • Young Adult

Substances

  • Antigens, CD
  • Apoptosis Regulatory Proteins
  • CD4 Antigens
  • IL2RA protein, human
  • Interleukin-2 Receptor alpha Subunit
  • PDCD1 protein, human
  • Programmed Cell Death 1 Receptor