The ever-expanding function of NOD2: autophagy, viral recognition, and T cell activation

Trends Immunol. 2011 Feb;32(2):73-9. doi: 10.1016/j.it.2010.12.007. Epub 2011 Jan 19.

Abstract

The identification of several families of innate pattern recognition receptors has greatly enhanced our understanding of the host innate immune response against a variety of pathogens. One such family of innate receptors is the nucleotide-binding domain and leucine rich repeat containing receptors (NLRs). NOD2 has been characterized as a cytosolic sensor of bacteria peptidoglycan (PGN). For almost 10 years, NOD2 was assigned with the function of mediating the RICK- and nuclear factor-κB induced proinflammatory response triggered by PGN. Recent studies have extended the biological activity of NOD2 to include the induction of autophagy and antiviral responses, as well as mediating direct T cell activation. Here, we highlight and discuss these new findings in the context of immune activation and pathogen detection.

Publication types

  • Research Support, N.I.H., Extramural
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adaptive Immunity
  • Animals
  • Autophagy*
  • Humans
  • Lymphocyte Activation*
  • Nod2 Signaling Adaptor Protein / genetics
  • Nod2 Signaling Adaptor Protein / immunology*
  • T-Lymphocytes / immunology*
  • T-Lymphocytes / virology*

Substances

  • Nod2 Signaling Adaptor Protein