Neuronal differentiation of C17.2 neural stem cells induced by a natural flavonoid, baicalin

Chembiochem. 2011 Feb 11;12(3):449-56. doi: 10.1002/cbic.201000570. Epub 2011 Jan 4.

Abstract

Natural medicinal materials are a significant resource for the identification of compounds with specific biological properties. In this study, we employed multipotent C17.2 neural stem cells as a model for screening natural compounds that possess neural differentiation-inducing properties. We show here for the first time that, out of the 67 compounds tested, the flavonoid baicalin is a potent differentiation-inducing agent. Baicalin increased the number of cells bearing extended neurites and the expression levels of a number of neuronal markers. Importantly, baicalin promoted the expression of several key neurogenic transcriptional factors. Moreover, we demonstrated that baicalin enhanced the phosphorylation/activation of Erk1/2. Inhibition of Erk1/2 activation by the MEK inhibitor U0126 attenuated the neuronal differentiation-inducing effect of baicalin. Taken together, our findings suggest that baicalin induces neuronal differentiation of C17.2 neural stem cells and that this is mediated by activation of Erk1/2. Our work lays the foundation for exploring baicalin for the promotion of neural regeneration after injury or disease.

MeSH terms

  • Animals
  • Butadienes / pharmacology
  • Cell Differentiation
  • Flavonoids / pharmacology*
  • Gene Expression Regulation
  • Mice
  • Mitogen-Activated Protein Kinase 1 / antagonists & inhibitors
  • Mitogen-Activated Protein Kinase 1 / metabolism
  • Mitogen-Activated Protein Kinase 3 / antagonists & inhibitors
  • Mitogen-Activated Protein Kinase 3 / metabolism
  • Nerve Growth Factors / genetics
  • Nerve Growth Factors / metabolism
  • Neural Stem Cells / cytology
  • Neural Stem Cells / drug effects*
  • Nitriles / pharmacology
  • Phosphorylation

Substances

  • Butadienes
  • Flavonoids
  • Nerve Growth Factors
  • Nitriles
  • U 0126
  • baicalin
  • Mitogen-Activated Protein Kinase 1
  • Mitogen-Activated Protein Kinase 3