Thyroid-stimulating hormone, 5-HTTLPR genotype, and antidepressant response in depressed women

Psychiatr Genet. 2011 Oct;21(5):253-6. doi: 10.1097/YPG.0b013e3283457be5.

Abstract

Basal serum thyroid-stimulating hormone (TSH) levels may predict antidepressant efficacy in patients with major depressive episodes (MDE), but data are inconsistent. As the SS genotype of the 5-HTTLPR polymorphism has been associated with a lower antidepressant efficacy in women with MDE, we aimed at assessing the relationship between normal basal TSH, 5-HTTLPR, and antidepressant efficacy in women. A total of 71 women and 28 men, with normal baseline TSH serum levels, hospitalized for a MDE, were assessed for 5-HTTLPR genotypes and prospectively followed for short-term antidepressant efficacy. Women with SS genotype had higher TSH levels (P=0.002) and a worse antidepressant response (P=0.046) than the women with LL/LS genotype, whereas no significant difference was shown in men. In multivariate analyses, antidepressant response in women was explained by TSH and 5-HTTLPR, but not by other variables. Further research is needed to understand the underlying mechanism explaining interactions between sex, TSH, and serotonergic function.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Antidepressive Agents / therapeutic use*
  • Depressive Disorder, Major / blood
  • Depressive Disorder, Major / drug therapy*
  • Depressive Disorder, Major / genetics*
  • Female
  • Genetic Predisposition to Disease*
  • Humans
  • Logistic Models
  • Male
  • Serotonin Plasma Membrane Transport Proteins / genetics*
  • Thyrotropin / blood*

Substances

  • Antidepressive Agents
  • SLC6A4 protein, human
  • Serotonin Plasma Membrane Transport Proteins
  • Thyrotropin