microRNA-145 suppresses lung adenocarcinoma-initiating cell proliferation by targeting OCT4

Oncol Rep. 2011 Jun;25(6):1747-54. doi: 10.3892/or.2011.1252. Epub 2011 Apr 7.

Abstract

Recent studies demonstrated that microRNA-145 is downregulated in human cancer cells and may function as a tumor suppressor. However, its role in lung cancer tumorigenesis remains unclear. Here, we demonstrated that upregulation of miR-145 reduced the proliferation and invasion as well as the ratio of CD133-positive initiating cells and the tumorosphere growth capacity of the human lung adenocarcinoma A549 cell line. The direct targeting of miR-145 to OCT4 mRNA was predicted by bioinformatic analysis and validated by a luciferase reporter system. We, therefore, confirmed that miR-145 can impair the proliferation of human lung adenocarcinoma-initiating cells by targeting OCT4 and leads to inhibition of lung cancer development.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adenocarcinoma / genetics*
  • Adenocarcinoma / metabolism
  • Adenocarcinoma / pathology
  • Blotting, Western
  • Cell Line, Tumor
  • Cell Movement
  • Cell Proliferation*
  • Humans
  • Lung Neoplasms / genetics*
  • Lung Neoplasms / metabolism
  • Lung Neoplasms / pathology
  • MicroRNAs / genetics*
  • MicroRNAs / metabolism
  • Octamer Transcription Factor-3 / genetics*
  • Octamer Transcription Factor-3 / metabolism
  • Reverse Transcriptase Polymerase Chain Reaction
  • Transfection

Substances

  • MIRN145 microRNA, human
  • MicroRNAs
  • Octamer Transcription Factor-3
  • POU5F1 protein, human