Rational design of an aryl-C-glycoside catalyst from a natural product O-glycosyltransferase

Chem Biol. 2011 Apr 22;18(4):520-30. doi: 10.1016/j.chembiol.2011.02.013.

Abstract

Because the sugar moieties of natural products are primarily O-linked, the hydrolytic sensitivity of the glycosidic linkage limits their therapeutic application. One potential solution to this problem is to replace the labile O-glycosidic bond with an enzymatically and chemically stable C-glycosidic bond. In this study, computational analysis of the O-glycosyltransferase LanGT2 and the C-glycosyltransferase UrdGT2 was used to predict the changes necessary to switch the O-glycosylating enzyme to a C-glycosyltransferase. By screening rationally designed LanGT2 mutants a number of LanGT2 variants with C-glycosyltransferase activity were identified. One variant, having 10 amino acid substitutions, revealed the primary region that determines O- versus C-glycosylation. By modeling the active site of this mutant and probing the role of active site residues with alanine substitutions, this work also illuminates the mechanistic features of O- and C-glycosylation.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Amino Acid Sequence
  • Aminoglycosides / biosynthesis
  • Biocatalysis*
  • Biological Products / chemistry
  • Biological Products / genetics*
  • Biological Products / metabolism*
  • Glycosides / chemistry
  • Glycosides / metabolism*
  • Glycosylation
  • Glycosyltransferases / chemistry
  • Glycosyltransferases / genetics*
  • Glycosyltransferases / metabolism*
  • Models, Molecular
  • Molecular Sequence Data
  • Protein Conformation
  • Protein Engineering / methods*
  • Recombinant Fusion Proteins / chemistry
  • Recombinant Fusion Proteins / genetics
  • Recombinant Fusion Proteins / metabolism
  • Streptomyces / enzymology
  • Streptomyces / genetics
  • Substrate Specificity

Substances

  • Aminoglycosides
  • Biological Products
  • Glycosides
  • Recombinant Fusion Proteins
  • landomycin A
  • Glycosyltransferases